Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Neurocognition in individuals at high familial risk of mood disorders with or without subsequent onset of depression.

Psychological medicine - 1 Nov 2015

Papmeyer M, Sussmann J E, Hall J, McKirdy J, Peel A, Macdonald A, Lawrie S M, Whalley H C, McIntosh A M

Abstract excerpt

BACKGROUND: Neurocognitive performance deficits have been observed in mood disorder patients and their unaffected relatives and may therefore qualify as endophenotypes. However, the precise time course of neurocognitive deficits has not been studied so that it is unknown whether neurocognitive abnormalities reflect the early effects of familial vulnerability to mood disorders or if they emerge at illness onset....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bipolar Disorder
Share to Cognition Disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Depressive Disorder, Major

Share to Endophenotypes

Share to Follow-Up Studies

Share to Genetic Predisposition to Disease

Share to Young Adult

Source

Open in PubMed Open full text

Identifiers

PMID: 26189425
DOI: 10.1017/S0033291715001324
PMCID: PMC5034888

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Neurocognition in individuals at high familial risk of mood disorders with or without subsequent onset of depression.PMID 26189425Read ↗

Impairment of executive function and attention predicts onset of affective disorder in healthy high-risk twins.Semantic proximity Read ↗

Impairment of executive function but not memory in first-degree relatives of patients with bipolar I disorder and in euthymic patients with unipolar depression.Semantic proximity Read ↗

Cognitive function in unaffected twins discordant for affective disorder.Semantic proximity Read ↗

Cognitive functioning and Anxiety/Depression: Accounting for Genetic Confounding in the ALSPAC Cohort.Semantic proximity Read ↗

Set shifting and inhibition deficits as potential endophenotypes for depressionSemantic proximity Read ↗

Cognitive functioning in patients with familial bipolar I disorder and their unaffected relatives.Semantic proximity Read ↗

Neurocognitive allied phenotypes for schizophrenia and bipolar disorder.Semantic proximity Read ↗

Neurocognition and its association with adverse childhood experiences and familial risk of mental illnessSemantic proximity Read ↗

Meta-analyses of cognitive functioning in euthymic bipolar patients and their first-degree relatives.Semantic proximity Read ↗

Cognitive endophenotypes of bipolar disorder: a meta-analysis of neuropsychological deficits in euthymic patients and their first-degree relatives.Semantic proximity Read ↗

Neuropsychological and social cognitive function in young people at genetic risk of bipolar disorder.Semantic proximity Read ↗

Cortical Thickness in Individuals at High Familial Risk of Mood Disorders as They Develop Major Depressive Disorder.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.