Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Of mothers and myelin: Aberrant myelination phenotypes in mouse model of Angelman syndrome are dependent on maternal and dietary influences.

Behavioural brain research - 15 Sept 2015

Grier Mark D, Carson Robert P, Lagrange Andre H

Abstract excerpt

Angelman syndrome (AS) is a neurodevelopmental disorder characterized by a number of neurological problems, including developmental delay, movement disorders, and epilepsy. AS results from the loss of UBE3A (an imprinted gene) expressed from the maternal chromosome in neurons. Given the ubiquitous expression of Ube3a and the devastating nature of AS, the role of environmental and maternal effects has been largely...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angelman Syndrome
Share to Cerebral Cortex

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Diet, High-Fat

Share to Maternal Nutritional Physiological Phenomena

Share to Myelin Proteins

Share to Receptors, Glucocorticoid

Share to Sciatic Nerve

Share to Spinal Cord

Source

Open in PubMed Open full text

Identifiers

PMID: 26028516
DOI: 10.1016/j.bbr.2015.05.045
PMCID: PMC4496262

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Of mothers and myelin: Aberrant myelination phenotypes in mouse model of Angelman syndrome are dependent on maternal and dietary influences.PMID 26028516Read ↗

Comparative profiling of white matter development in the human and mouse brain reveals volumetric deficits and delayed myelination in Angelman syndromeSemantic proximity Read ↗

Angelman syndrome: insights into genomic imprinting and neurodevelopmental phenotypes.Semantic proximity Read ↗

Comparative profiling of white matter development in the human and mouse brain reveals volumetric deficits and delayed myelination in Angelman syndromeSemantic proximity Read ↗

Behavioral deficits in an Angelman syndrome model: effects of genetic background and age.Semantic proximity Read ↗

Genome-wide gene expression profiling of the Angelman syndrome mice with Ube3a mutation.Semantic proximity Read ↗

Angelman Syndrome.Semantic proximity Read ↗

Dietary Intake of Octanoic Acid Restores UBE3A Expression and Improves the Behavioral Phenotypes in a Mouse Model of Angelman SyndromeSemantic proximity Read ↗

Angelman Syndrome: From Mouse Models to Therapy.Semantic proximity Read ↗

Angelman syndrome: Mutations influence features in early childhood.Semantic proximity Read ↗

Neurobehavioral and electroencephalographic abnormalities in Ube3a maternal-deficient mice.Semantic proximity Read ↗

Assessing the requirements of prenatal UBE3A expression for rescue of behavioral phenotypes in a mouse model for Angelman syndrome.Semantic proximity Read ↗

Toward a Broader View of Ube3a in a Mouse Model of Angelman Syndrome: Expression in Brain, Spinal Cord, Sciatic Nerve and Glial Cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.