Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

CHOP deficiency prevents methylglyoxal-induced myocyte apoptosis and cardiac dysfunction

29 May 2015

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Advanced Glycation End Products research
Share to Autophagy in Disease and Therapy
Share to Endoplasmic Reticulum Stress and Disease

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 26027784
DOI: 10.1016/j.yjmcc.2015.05.016

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 24 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

CHOP deficiency prevents methylglyoxal-induced myocyte apoptosis and cardiac dysfunctionPMID 26027784Read ↗

Regulation of cardiac myocyte cell death and differentiation by myocardinSemantic proximity Read ↗

Cardiomyocyte apoptosis in the failing heart — A critical review from definition and classification of cell deathSemantic proximity Read ↗

Diabetes Promotes Cardiac Stem Cell Aging and Heart Failure, Which Are Prevented by Deletion of the p66 <sup>shc</sup> GeneSemantic proximity Read ↗

Methylglyoxal Induces Endothelial Dysfunction via Stunning-like PhenotypeSemantic proximity Read ↗

Novel insights in pathophysiology of antiblastic drugs-induced cardiotoxicity and cardioprotection.Semantic proximity Read ↗

Cardiac effects and clinical applications of MG53Semantic proximity Read ↗

The Pathogenic Role of the DNA Double-Stranded Breaks in Hereditary Cardiomyopathies.Semantic proximity Read ↗

Transcription Factor C/EBP Homologous Protein in Health and DiseasesSemantic proximity Read ↗

Myocyte apoptosis in heart failureSemantic proximity Read ↗

Identification of Protein Disulfide Isomerase as a Cardiomyocyte Survival Factor in Ischemic CardiomyopathySemantic proximity Read ↗

m6A demethylase ALKBH5 attenuates doxorubicin-induced cardiotoxicity via posttranscriptional stabilization of Rasal3Semantic proximity Read ↗

IDH2 deficiency exacerbates acetaminophen hepatotoxicity in mice via mitochondrial dysfunction-induced apoptosisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.