A genome-wide association study identifies PLCL2 and AP3D1-DOT1L-SF3A2 as new susceptibility loci for myocardial infarction in Japanese.

doi:10.1038/ejhg.2014.110

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

A genome-wide association study identifies PLCL2 and AP3D1-DOT1L-SF3A2 as new susceptibility loci for myocardial infarction in Japanese.

European journal of human genetics : EJHG - 1 Mar 2015

Hirokawa Megumi, Morita Hiroyuki, Tajima Tomoyuki, Takahashi Atsushi, Ashikawa Kyota, Miya Fuyuki, Shigemizu Daichi, Ozaki Kouichi, Sakata Yasuhiko, Nakatani Daisaku, Suna Shinichiro, Imai Yasushi, Tanaka Toshihiro, Tsunoda Tatsuhiko, Matsuda Koichi, Kadowaki Takashi, Nakamura Yusuke, Nagai Ryozo, Komuro Issei, Kubo Michiaki

Abstract excerpt

Despite considerable progress in preventive and therapeutic strategies, myocardial infarction (MI) is one of the leading causes of death throughout the world. A total of 55 susceptibility genes have been identified mostly in European genome-wide association studies (GWAS). Nevertheless, large-scale GWAS from other population could possibly find additional susceptibility loci. To identify as many MI susceptibility...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromosomes, Human, Pair 12

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gene Regulatory Networks

Share to Histone-Lysine N-Methyltransferase

Share to Intracellular Signaling Peptides and Proteins

Share to Methyltransferases

Share to Myocardial Infarction

Share to RNA-Binding Proteins

Share to RNA Splicing Factors

Source

Open in PubMed Open full text

Identifiers

PMID: 24916648
DOI: 10.1038/ejhg.2014.110
PMCID: PMC4326706

Genes

PLCL2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A genome-wide association study identifies PLCL2 and AP3D1-DOT1L-SF3A2 as new susceptibility loci for myocardial infarction in Japanese.PMID 24916648Read ↗

Molecular genetics of coronary artery disease.Semantic proximity Read ↗

Identification of STXBP2 as a novel susceptibility locus for myocardial infarction in Japanese individuals by an exome-wide association study.Semantic proximity Read ↗

Association of a polymorphism of BTN2A1 with myocardial infarction in East Asian populations.Semantic proximity Read ↗

Genetic variants associated with myocardial infarction and the risk factors in Chinese population.Semantic proximity Read ↗

The discovery of genes implicated in myocardial infarction.Semantic proximity Read ↗

Association of six genetic variants with myocardial infarction.Semantic proximity Read ↗

Association of genetic variants in SEMA3F, CLEC16A, LAMA3, and PCSK2 with myocardial infarction in Japanese individuals.Semantic proximity Read ↗

Large scale association analysis identifies three susceptibility loci for coronary artery disease.Semantic proximity Read ↗

Genome-wide association study of coronary artery disease in the Japanese.Semantic proximity Read ↗

Analysis of 17,576 potentially functional SNPs in three case-control studies of myocardial infarction.Semantic proximity Read ↗

ACSL1, CH25H, GPCPD1, and PLA2G12A as the potential lipid-related diagnostic biomarkers of acute myocardial infarctionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.