Evidences of +896 A/G TLR4 polymorphism as an indicative of prevalence of complications in T2DM patients.

doi:10.1155/2014/973139

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Evidences of +896 A/G TLR4 polymorphism as an indicative of prevalence of complications in T2DM patients.

Mediators of inflammation - 1 Jan 2014

Balistreri Carmela Rita, Bonfigli Anna Rita, Boemi Massimo, Olivieri Fabiola, Ceriello Antonio, Genovese Stefano, Franceschi Claudio, Spazzafumo Liana, Fabietti Paolo, Candore Giuseppina, Caruso Calogero, Lio Domenico, Testa Roberto

Abstract excerpt

T2DM is today considered as world-wide health problem, with complications responsible of an enhanced mortality and morbidity. Thus, new strategies for its prevention and therapy are necessary. For this reason, the research interest has focused its attention on TLR4 and its polymorphisms, particularly the rs4986790. However, no conclusive findings have been reported until now about the role of this polymorphism in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aged

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Aged, 80 and over

Share to Diabetes Mellitus, Type 2

Share to Gene Frequency

Share to Genetic Predisposition to Disease

Share to Genotype

Share to Polymorphism, Single Nucleotide

Share to Toll-Like Receptor 4

Source

Open in PubMed Open full text

Identifiers

PMID: 24803744
DOI: 10.1155/2014/973139
PMCID: PMC3996297

Genes

TLR4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Evidences of +896 A/G TLR4 polymorphism as an indicative of prevalence of complications in T2DM patients.PMID 24803744Read ↗

Quantitative assessment of TLR4 gene polymorphisms and T2DM risk: A meta-analysis.Semantic proximity Read ↗

The effect of Toll-like receptor 4 gene polymorphism on vascular complications in type 2 diabetes patients.Semantic proximity Read ↗

Role of variants rs5030717 and rs5030718 of TLR4 in the risk prediction of nephropathy, hypertension and dyslipidaemia in type 2 diabetes mellitus.Semantic proximity Read ↗

TLR4 polymorphisms seem not to be associated with prediabetes and type 2 diabetes but predispose to diabetic retinopathy; TLR4 polymorphisms in glucose continuum.Semantic proximity Read ↗

Genetic associated complications of type 2 diabetes mellitus.Semantic proximity Read ↗

Association of toll-like receptor 4 polymorphisms with type 2 diabetes mellitus.Semantic proximity Read ↗

The roles of endothelial nitric oxide synthase gene polymorphisms in diabetes mellitus and its associated vascular complications: a systematic review and meta-analysis.Semantic proximity Read ↗

Mediators of diabetic neuropathy: is hyperglycemia the only culprit?Semantic proximity Read ↗

Toll-like receptors-2 and -9 (TLR2 and TLR9) gene polymorphism in patients with type 2 diabetes and diabetic foot.Semantic proximity Read ↗

Association between Genetic Variants and Diabetes Mellitus in Iranian Populations: A Systematic Review of Observational Studies.Semantic proximity Read ↗

Prevalence, patterns, and determinants of vascular complications of type 2 diabetes in a teaching hospital in Addis Ababa, Ethiopia: a cross-sectional studySemantic proximity Read ↗

Genetic Variants and Their Associations to Type 2 Diabetes Mellitus Complications in the United Arab Emirates.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.