Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Silencing of DS2 aminoacylase-like genes confirms basal resistance to Phytophthora infestans in Nicotiana benthamiana.

Plant signaling & behavior - 1 Jan 2014

Nakano Masahito, Nishihara Masahiro, Yoshioka Hirofumi, Ohnishi Kouhei, Hikichi Yasufumi, Kiba Akinori

Abstract excerpt

Nicotiana benthamiana is a potential host to several plant pathogens, and immature leaves of N. benthamiana are susceptible to Phytophthora infestans. In contrast, mature leaves of N. benthamiana are weakly susceptible and show basal resistance to P. infestans. We screened a gene-silenced mature plant showing high resistance to P. infestans, designated as DS2 (Disease suppression 2). The deduced amino acid...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amidohydrolases
Share to Disease Resistance

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Gene Silencing

Share to MAP Kinase Signaling System

Share to Nicotiana

Share to Phytophthora infestans

Share to Plant Immunity

Share to Receptors, Pattern Recognition

Source

Open in PubMed Open full text

Identifiers

PMID: 24514749
DOI: 10.4161/psb.28004
PMCID: PMC4091584

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Silencing of DS2 aminoacylase-like genes confirms basal resistance to Phytophthora infestans in Nicotiana benthamiana.PMID 24514749Read ↗

Resistance to Phytophthora infestans: exploring genes required for disease resistance in Solanaceae plantsSemantic proximity Read ↗

Phytosterols play a key role in plant innate immunity against bacterial pathogens by regulating nutrient efflux into the apoplast.Semantic proximity Read ↗

Activation tagging of ADR2 conveys a spreading lesion phenotype and resistance to biotrophic pathogens.Semantic proximity Read ↗

The Arabidopsis aberrant growth and death2 mutant shows resistance to Pseudomonas syringae and reveals a role for NPR1 in suppressing hypersensitive cell death.Semantic proximity Read ↗

SGT1 and HSP90 are essential for age-related non-host resistance of Nicotiana benthamiana against the oomycete pathogen Phytophthora infestansSemantic proximity Read ↗

Divergent roles in Arabidopsis thaliana development and defense of two homologous genes, aberrant growth and death2 and AGD2-LIKE DEFENSE RESPONSE PROTEIN1, encoding novel aminotransferases.Semantic proximity Read ↗

Two Chloroplast-Localized Proteins: AtNHR2A and AtNHR2B, Contribute to Callose Deposition During Nonhost Disease Resistance in Arabidopsis.Semantic proximity Read ↗

Programmed cell death in plants: a pathogen-triggered response activated coordinately with multiple defense functions.Semantic proximity Read ↗

The Arabidopsis thaliana dihydroxyacetone phosphate reductase gene SUPPRESSSOR OF FATTY ACID DESATURASE DEFICIENCY1 is required for glycerolipid metabolism and for the activation of systemic acquired resistance.Semantic proximity Read ↗

Immunity-Associated Programmed Cell Death as a Tool for the Identification of Genes Essential for Plant Innate Immunity.Semantic proximity Read ↗

Abscisic acid has a key role in modulating diverse plant-pathogen interactions.Semantic proximity Read ↗

Knocking down expression of the auxin-amidohydrolase IAR3 alters defense responses in Solanaceae family plants.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.