Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

NADPH Oxidase, NOX1, Mediates Vascular Injury in Ischemic Retinopathy

22 Sept 2013

Abstract excerpt

AIMS: Ischemic retinal diseases such as retinopathy of prematurity are major causes of blindness due to damage to the retinal microvasculature. Despite this clinical situation, retinopathy of prematurity is mechanistically poorly understood. Therefore, effective preventative therapies are not available. However, hypoxic-induced increases in reactive oxygen species (ROS) have been suggested to be involved with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Neutrophil, Myeloperoxidase and Oxidative Mechanisms
Share to Retinal Development and Disorders
Share to Retinopathy of Prematurity Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24053718
DOI: 10.1089/ars.2013.5357

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

NADPH Oxidase, NOX1, Mediates Vascular Injury in Ischemic RetinopathyPMID 24053718Read ↗

Animal Models of Retinopathy of Prematurity: Advances and Metabolic RegulatorsSemantic proximity Read ↗

Retinopathy of Prematurity—Targeting Hypoxic and Redox Signaling PathwaysSemantic proximity Read ↗

Vascular Endothelial Growth Factor Signaling in Models of Oxygen-Induced Retinopathy: Insights Into Mechanisms of Pathology in Retinopathy of PrematuritySemantic proximity Read ↗

Retinopathy of Prematurity: Current Concepts in Molecular PathogenesisSemantic proximity Read ↗

Galectin-1 expression imprints a neurovascular phenotype in proliferative retinopathies and delineates responses to anti-VEGF.Semantic proximity Read ↗

Lack of the Antioxidant Glutathione Peroxidase-1 (GPx1) Exacerbates Retinopathy of Prematurity in MiceSemantic proximity Read ↗

Roles of Hypoxia Response in Retinal Development and PathophysiologySemantic proximity Read ↗

Retinopathy of prematurity: from oxygen management to molecular manipulationSemantic proximity Read ↗

Atypical p38 Kinase Signaling in Retinal Vascular Damage and RecoverySemantic proximity Read ↗

Requirement of NOX2 expression in both retina and bone marrow for diabetes-induced retinal vascular injury.Semantic proximity Read ↗

Detailed investigation on the role of lipid metabolizing enzymes in the pathogenesis of retinopathy of prematurity among preterm infantsSemantic proximity Read ↗

Angiogenesis and Eye DiseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.