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Article

FUS-mediated alternative splicing in the nervous system: consequences for ALS and FTLD

23 Aug 2013

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23974990
DOI: 10.1007/s00109-013-1077-2

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FUS-mediated alternative splicing in the nervous system: consequences for ALS and FTLDPMID 23974990Read ↗

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The splice of life: Alternative splicing and neurological diseaseSemantic proximity Read ↗

Proteomic analysis of FUS interacting proteins provides insights into FUS function and its role in ALSSemantic proximity Read ↗

Alternative Splicing of ALS Genes: Misregulation and Potential TherapiesSemantic proximity Read ↗

FUS-regulated region- and cell-type-specific transcriptome is associated with cell selectivity in ALS/FTLDSemantic proximity Read ↗

The minor spliceosome could be the major key for FUS/TLS mutants in ALS.Semantic proximity Read ↗

Splicing Regulation in Neurologic DiseaseSemantic proximity Read ↗

Tau gene alternative splicing: expression patterns, regulation and modulation of function in normal brain and neurodegenerative diseasesSemantic proximity Read ↗

FUS gene mutations associated with familiar forms of amyotrophic lateral sclerosis affect cellular localization and aggregation properties of the encoded protein.Semantic proximity Read ↗

Pathogenesis of FUS-associated ALS and FTD: insights from rodent modelsSemantic proximity Read ↗

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