Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Helicobacter pylori induces type 4 secretion system-dependent, but CagA-independent activation of IκBs and NF-κB/RelA at early time points

26 Jul 2013

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Galectins and Cancer Biology
Share to Helicobacter pylori-related gastroenterology studies
Share to Veterinary medicine and infectious diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 23972614
DOI: 10.1016/j.ijmm.2013.07.008

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Helicobacter pylori induces type 4 secretion system-dependent, but CagA-independent activation of IκBs and NF-κB/RelA at early time pointsPMID 23972614Read ↗

Interplay of the Gastric Pathogen<i>Helicobacter pylori</i>with Toll-Like ReceptorsSemantic proximity Read ↗

Investigating the Molecular Mechanisms Underlying Early Response to Inflammation and Helicobacter pylori Infection in Human Gastric Epithelial CellsSemantic proximity Read ↗

What a disorder: proinflammatory signaling pathways induced by Helicobacter pyloriSemantic proximity Read ↗

The Importance of Toll‐like Receptors in NF ‐κB Signaling Pathway Activation by Helicobacter pylori Infection and the Regulators of this ResponseSemantic proximity Read ↗

Inflammation and proliferation – a causal event of host response to Helicobacter pylori infectionSemantic proximity Read ↗

TIFA Signaling in Gastric Epithelial Cells Initiates the cag Type 4 Secretion System-Dependent Innate Immune Response to Helicobacter pylori InfectionSemantic proximity Read ↗

Macrophages recognize the Helicobacter pylori type IV secretion system in the absence of toll-like receptor signallingSemantic proximity Read ↗

Helicobacter pylori actively suppresses innate immune nucleic acid receptorsSemantic proximity Read ↗

Activation of IL-8 Gene Expression by <i>Helicobacter pylori</i> Is Regulated by Transcription Factor Nuclear Factor-κB in Gastric Epithelial CellsSemantic proximity Read ↗

Innate immune activation and modulatory factors of Helicobacter pylori towards phagocytic and nonphagocytic cellsSemantic proximity Read ↗

Helicobacter pylori CagA activates NF‐κB by targeting TAK1 for TRAF6‐mediated Lys 63 ubiquitinationSemantic proximity Read ↗

NF‐κB Signaling in Gastric CancerSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.