Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Two patients with history of STEC-HUS, posttransplant recurrence and complement gene mutations.

American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons - 1 Aug 2013

Alberti M, Valoti E, Piras R, Bresin E, Galbusera M, Tripodo C, Thaiss F, Remuzzi G, Noris M

Abstract excerpt

Hemolytic uremic syndrome (HUS) is a disease of microangiopathic hemolytic anemia, thrombocytopenia and acute renal failure. About 90% of cases are secondary to infections by Escherichia coli strains producing Shiga-like toxins (STEC-HUS), while 10% are associated with mutations in genes encoding...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Complement Factor I
Share to Escherichia coli Infections

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Graft Rejection

Share to Hemolytic-Uremic Syndrome

Share to Kidney Failure, Chronic

Share to Membrane Cofactor Protein

Share to Shiga-Toxigenic Escherichia coli

Share to Thrombocytopenia

Source

Identifiers

PMID: 23731345
DOI: 10.1111/ajt.12297

Genes

CFI

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-23731345-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Two patients with history of STEC-HUS, posttransplant recurrence and complement gene mutations.PMID 23731345Read ↗

Posttransplant recurrence of atypical hemolytic uremic syndrome.Semantic proximity Read ↗

Novel Complement Factor B Gene Mutation Identified in a Kidney Transplant Recipient with a Shiga Toxin-Triggered Episode of Thrombotic Microangiopathy.Semantic proximity Read ↗

Outcome of renal transplantation in patients with non-Shiga toxin-associated hemolytic uremic syndrome: prognostic significance of genetic background.Semantic proximity Read ↗

HUS with mutations in CFH and STEC infection treated with eculizumab in a 4-year-old girl.Semantic proximity Read ↗

A newly identified mutation in the complement factor I gene not associated with early post-transplant recurrence of atypical hemolytic-uremic syndrome: a case report.Semantic proximity Read ↗

Hemolytic uremic syndrome with simultaneous Shiga toxin producing Escherichia coli and complement abnormalities.Semantic proximity Read ↗

Recurrence of hemolytic uremic syndrome after renal transplantation.Semantic proximity Read ↗

Thrombotic microangiopathy after kidney transplantation.Semantic proximity Read ↗

Complement genes strongly predict recurrence and graft outcome in adult renal transplant recipients with atypical hemolytic and uremic syndrome.Semantic proximity Read ↗

Shiga toxin-producing Escherichia coli in household members of children with hemolytic uremic syndrome.Semantic proximity Read ↗

CFH gene mutation in a case of Shiga toxin-associated hemolytic uremic syndrome (STEC-HUS).Semantic proximity Read ↗

Genetic Susceptibility to Postdiarrheal Hemolytic-Uremic Syndrome After Shiga Toxin-Producing Escherichia coli Infection: A Centers for Disease Control and Prevention FoodNet Study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.