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Article

Mitochondrial quality control turns out to be the principal suspect in parkin and PINK1-related autosomal recessive Parkinson's disease

30 Nov 2012

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Share to Mitochondrial Function and Pathology
Share to Neurological diseases and metabolism
Share to Parkinson's Disease Mechanisms and Treatments

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23206589
DOI: 10.1016/j.conb.2012.11.002

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Related research

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Mitochondrial quality control turns out to be the principal suspect in parkin and PINK1-related autosomal recessive Parkinson's diseasePMID 23206589Read ↗

The PINK1/Parkin pathway: a mitochondrial quality control system?Semantic proximity Read ↗

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Mitochondrial Quality Control and Parkinson’s Disease: A Pathway UnfoldsSemantic proximity Read ↗

Parkin and mitochondrial quality control: toward assembling the puzzleSemantic proximity Read ↗

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PINK1, Parkin, and Mitochondrial Quality Control: What can we Learn about Parkinson’s Disease Pathobiology?Semantic proximity Read ↗

The Roles of PINK1, Parkin, and Mitochondrial Fidelity in Parkinson’s DiseaseSemantic proximity Read ↗

Parkin and PINK1 function in a vesicular trafficking pathway regulating mitochondrial quality controlSemantic proximity Read ↗

Parkin, an E3 Ubiquitin Ligase, Plays an Essential Role in Mitochondrial Quality Control in Parkinson's Disease.Semantic proximity Read ↗

Mitochondrial quality control in neurodegenerative diseases.Semantic proximity Read ↗

Mitochondrial quality control: insights on how Parkinson's disease related genes PINK1, parkin, and Omi/HtrA2 interact to maintain mitochondrial homeostasis.Semantic proximity Read ↗

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