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Article

A molecular mechanism underlying ovarian dysfunction of polycystic ovary syndrome: hyperandrogenism induces epigenetic alterations in the granulosa cells

20 Feb 2012

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Share to Ovarian cancer diagnosis and treatment
Share to Ovarian function and disorders
Share to Reproductive Biology and Fertility

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22349439
DOI: 10.1007/s00109-012-0881-4

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A molecular mechanism underlying ovarian dysfunction of polycystic ovary syndrome: hyperandrogenism induces epigenetic alterations in the granulosa cellsPMID 22349439Read ↗

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Altered miR-186 and miR-135a contribute to granulosa cell dysfunction by targeting ESR2: A possible role in polycystic ovary syndromeSemantic proximity Read ↗

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The Role of microRNAs in Ovarian Granulosa Cells in Health and DiseaseSemantic proximity Read ↗

Downregulation of lncRNA ZFAS1 and upregulation of microRNA-129 repress endocrine disturbance, increase proliferation and inhibit apoptosis of ovarian granulosa cells in polycystic ovarian syndrome by downregulating HMGB1Semantic proximity Read ↗

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