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Article

PINK1 and Parkin Target Miro for Phosphorylation and Degradation to Arrest Mitochondrial Motility

1 Nov 2011

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Share to Autophagy in Disease and Therapy
Share to Mitochondrial Function and Pathology
Share to Parkinson's Disease Mechanisms and Treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 22078885
DOI: 10.1016/j.cell.2011.10.018

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

PINK1 and Parkin Target Miro for Phosphorylation and Degradation to Arrest Mitochondrial MotilityPMID 22078885Read ↗

PINK1 and Parkin Flag Miro to Direct Mitochondrial TrafficSemantic proximity Read ↗

Miro phosphorylation sites regulate Parkin recruitment and mitochondrial motility.Semantic proximity Read ↗

Deciphering the Molecular Signals of PINK1/Parkin MitophagySemantic proximity Read ↗

Parkin and PINK1 function in a vesicular trafficking pathway regulating mitochondrial quality controlSemantic proximity Read ↗

Functional Impairment in Miro Degradation and Mitophagy Is a Shared Feature in Familial and Sporadic Parkinson's Disease.Semantic proximity Read ↗

The Role of PTEN-L in Modulating PINK1-Parkin-Mediated MitophagySemantic proximity Read ↗

Parkin and PINK1: much more than mitophagySemantic proximity Read ↗

p32 regulates mitochondrial morphology and dynamics through parkinSemantic proximity Read ↗

PINK1/Parkin-mediated mitophagy in mammalian cellsSemantic proximity Read ↗

PINK1/Parkin-mediated mitophagy in neurodegenerative diseasesSemantic proximity Read ↗

Mechanisms of neurodegeneration in Parkinson’s disease: keep neurons in the PINK1Semantic proximity Read ↗

Parkin ubiquitinates mTOR to regulate mTORC1 activity under mitochondrial stressSemantic proximity Read ↗

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