Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

<i>Chlamydia trachomatis</i> Infection Causes Mitotic Spindle Pole Defects Independently from its Effects on Centrosome Amplification

7 Apr 2011

Abstract excerpt

Chlamydiae are Gram negative, obligate intracellular bacteria, and Chlamydia trachomatis is the etiologic agent of the most commonly reported sexually transmitted disease in the United States. Chlamydiae undergo a biphasic life cycle that takes place inside a parasitophorous vacuole termed an inclusion. Chlamydial infections have been epidemiologically linked to cervical cancer in patients previously infected by...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Microtubule and mitosis dynamics
Share to Ovarian cancer diagnosis and treatment
Share to Reproductive Biology and Fertility

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 21477082
DOI: 10.1111/j.1600-0854.2011.01204.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

<i>Chlamydia trachomatis</i> Infection Causes Mitotic Spindle Pole Defects Independently from its Effects on Centrosome AmplificationPMID 21477082Read ↗

Chlamydia trachomatis Causes Centrosomal Defects Resulting in Chromosomal Segregation AbnormalitiesSemantic proximity Read ↗

Chlamydia and HPV induce centrosome amplification in the host cell through additive mechanismsSemantic proximity Read ↗

Multinucleation during C. trachomatis infections is caused by the contribution of two effector pathways.Semantic proximity Read ↗

A renewed tool kit to explore Chlamydia pathogenesis: from molecular genetics to new infection modelsSemantic proximity Read ↗

Taking control: reorganization of the host cytoskeleton by ChlamydiaSemantic proximity Read ↗

Pathogenic Puppetry: Manipulation of the Host Actin Cytoskeleton by Chlamydia trachomatisSemantic proximity Read ↗

Separate To Operate: the Centriole-Free Inner Core of the Centrosome Regulates the Assembly of the Intranuclear Spindle in Toxoplasma gondiiSemantic proximity Read ↗

A Distinct Interphase Microtubule State Marks Host Cell Permissiveness to <i>Chlamydia pneumoniae</i> EntrySemantic proximity Read ↗

Biochemical and structural insights into microtubule perturbation by CopN from Chlamydia pneumoniae.Semantic proximity Read ↗

Cytokinesis is blocked in mammalian cells transfected with Chlamydia trachomatis gene CT223.Semantic proximity Read ↗

Polarized Cell Division of Chlamydia trachomatisSemantic proximity Read ↗

The <i>Chlamydia</i> protein CpoS modulates the inclusion microenvironment and restricts the interferon response by acting on Rab35Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.