Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Salmonella enterica Serovar Enteritidis tatB and tatC Mutants Are Impaired in Caco-2 Cell Invasion In Vitro and Show Reduced Systemic Spread in Chickens

25 May 2010

Abstract excerpt

Salmonella enterica subsp. enterica serovar Enteritidis is a leading causative agent of gastroenteritis in humans. This pathogen also colonizes the intestinal tracts of poultry and can spread systemically in chickens. Transfer to humans usually occurs through undercooked or improperly handled poultry meat or eggs. The bacterial twin-arginine transport (Tat) pathway is responsible for the translocation of folded...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacteriophages and microbial interactions
Share to Salmonella and Campylobacter epidemiology
Share to Viral gastroenteritis research and epidemiology

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 20498258
DOI: 10.1128/iai.00090-10

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Salmonella enterica Serovar Enteritidis tatB and tatC Mutants Are Impaired in Caco-2 Cell Invasion In Vitro and Show Reduced Systemic Spread in ChickensPMID 20498258Read ↗

Functional Characterization of the Twin-Arginine Translocation System in <i>Campylobacter jejuni</i>Semantic proximity Read ↗

Abrogation of the twin arginine transport system in Salmonella enterica serovar Typhimurium leads to colonization defects during infection.Semantic proximity Read ↗

Relationship Between the Tat Protein Transport System and Virulence in ETECSemantic proximity Read ↗

Twin-arginine translocation system (tat) mutants of Salmonella are attenuated due to envelope defects, not respiratory defects.Semantic proximity Read ↗

Relationship Between the Tat Protein Transport System and Virulence of Enterotoxigenic Escherichia ColiSemantic proximity Read ↗

Escherichia coli type III secretion system 2 regulator EtrA promotes virulence of avian pathogenic Escherichia coli.Semantic proximity Read ↗

The Twin Arginine Translocation System Is Essential for Virulence ofYersinia pseudotuberculosisSemantic proximity Read ↗

In vivo dissection of the Tat translocation pathway in Escherichia coli.Semantic proximity Read ↗

The role of the twin-arginine translocation pathway in Escherichia coli K1 pathogenicity in the African migratory locust, Locusta migratoria.Semantic proximity Read ↗

Proteomic analysis of a twin-arginine translocation-deficient mutant unravel its functions involved in stress adaptation and virulence in fish pathogen Edwardsiella tarda.Semantic proximity Read ↗

Salmonella enteritidis Effector AvrA Stabilizes Intestinal Tight Junctions via the JNK Pathway.Semantic proximity Read ↗

Twin arginine translocation (Tat)-dependent export in the apparent absence of TatABC or TatA complexes using modified Escherichia coli TatA subunits that substitute for TatB.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.