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Article

Three amino acid changes in PB1-F2 of highly pathogenic H5N1 avian influenza virus affect pathogenicity in mallard ducks

9 Apr 2010

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20383540
DOI: 10.1007/s00705-010-0666-4

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Related research

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Three amino acid changes in PB1-F2 of highly pathogenic H5N1 avian influenza virus affect pathogenicity in mallard ducksPMID 20383540Read ↗

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PB2 substitutions V598T/I increase the virulence of H7N9 influenza A virus in mammalsSemantic proximity Read ↗

Characterization of duck H5N1 influenza viruses with differing pathogenicity in mallard (<i>Anas platyrhynchos</i>) ducksSemantic proximity Read ↗

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A nonpathogenic duck-origin H9N2 influenza A virus adapts to high pathogenicity in miceSemantic proximity Read ↗

Differential Contribution of PB1-F2 to the Virulence of Highly Pathogenic H5N1 Influenza A Virus in Mammalian and Avian SpeciesSemantic proximity Read ↗

H5N1 high pathogenicity avian influenza virus in migratory birds exhibiting low pathogenicity in mallards increases its risk of transmission and spread in poultrySemantic proximity Read ↗

PB2-Q591K Mutation Determines the Pathogenicity of Avian H9N2 Influenza Viruses for Mammalian SpeciesSemantic proximity Read ↗

A single substitution in amino acid 184 of the NP protein alters the replication and pathogenicity of H5N1 avian influenza viruses in chickensSemantic proximity Read ↗

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