Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

T-786C polymorphism in the endothelial nitric oxide synthase gene is associated with increased risk of coronary artery disease in a Chinese population.

Pharmacology - 1 Jan 2010

Han Yi, Xu Weiwei, Zhang Wen, Liu Naifeng, Ji Yong

Abstract excerpt

BACKGROUND/AIMS: Many investigations have revealed that genetic influences can have an important impact on the incidence and progression of coronary artery disease (CAD). The aim of this research was to discover whether the T-786C polymorphism in the promoter 5'-flanking region of the gene for endothelial nitric oxide synthase (eNOS) is associated with CAD. METHODS: 622 Chinese Han subjects were enrolled in this...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Body Mass Index
Share to Cholesterol, LDL

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Coronary Artery Disease

Share to Hypertension

Share to Logistic Models

Share to Nitric Oxide Synthase Type III

Share to Sex Factors

Share to Smoking

Source

Identifiers

PMID: 20215811
DOI: 10.1159/000275135

Genes

CAD

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

T-786C polymorphism in the endothelial nitric oxide synthase gene is associated with increased risk of coronary artery disease in a Chinese population.PMID 20215811Read ↗

Association between the - 786T>C 1polymorphism in the promoter region of endothelial nitric oxide synthase (eNOS) and risk of coronary artery disease: a systematic review and meta-analysis.Semantic proximity Read ↗

The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study.Semantic proximity Read ↗

The relationship between endothelial nitric oxide synthase gene polymorphism (T-786 C) and coronary artery disease in the Turkish population.Semantic proximity Read ↗

Endothelial nitric oxide synthase gene polymorphisms and risk of coronary artery disease.Semantic proximity Read ↗

Association of endothelial nitric oxide synthase promoter region (T-786C) gene polymorphism with acute coronary syndrome and coronary heart disease.Semantic proximity Read ↗

The Endothelial Nitric Oxide Synthase Gene T-786C Polymorphism Increases Myocardial Infarction Risk: A Meta-Analysis.Semantic proximity Read ↗

The endothelial nitric oxide synthase gene is associated with coronary artery disease: a meta-analysis.Semantic proximity Read ↗

Association between eNOS polymorphisms and risk of coronary artery disease in a Korean population: a meta-analysis.Semantic proximity Read ↗

Association between endothelial nitric oxide synthase gene polymorphism (T-786C) and ischemic stroke susceptibility: a meta-analysis.Semantic proximity Read ↗

A T-786C polymorphism in 5'-flanking region of the endothelial nitric oxide synthase gene and endothelium-dependent arterial dilation in Type 2 diabetes.Semantic proximity Read ↗

Smoking-dependent and haplotype-specific effects of endothelial nitric oxide synthase gene polymorphisms on angiographically assessed coronary artery disease in Caucasian- and African-Brazilians.Semantic proximity Read ↗

Association of the Endothelial Nitric Oxide Synthase Gene T786C Polymorphism with In-Stent Restenosis in Chinese Han Patients with Coronary Artery Disease Treated with Drug-Eluting Stent.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.