Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Expression of transient receptor potential channel mucolipin (TRPML) and polycystine (TRPP) in the mouse inner ear.

Acta oto-laryngologica - 1 Feb 2010

Takumida Masaya, Anniko Matti

Abstract excerpt

CONCLUSIONS: TRPML3 may play distinct roles in the inner ear, such as stereociliar organization, sensory cell transduction, and inner ear fluid homeostasis, and TRPP3 may be important for fluid homeostasis in the inner ear. OBJECTIVE: To study the expression of TRPML1-3 and TRPP2, 3, and 5 in the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cochlea
Share to Ear, Inner
Share to Hair Cells, Auditory, Inner

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Hair Cells, Auditory, Outer

Share to Organ of Corti

Share to Transient Receptor Potential Channels

Share to TRPM Cation Channels

Share to TRPP Cation Channels

Source

Identifiers

PMID: 20095091
DOI: 10.3109/00016480903013593

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-20095091-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Expression of transient receptor potential channel mucolipin (TRPML) and polycystine (TRPP) in the mouse inner ear.PMID 20095091Read ↗

TRPML3 and hearing loss in the varitint-waddler mouse.Semantic proximity Read ↗

TRPML3 mutations cause impaired mechano-electrical transduction and depolarization by an inward-rectifier cation current in auditory hair cells of varitint-waddler mice.Semantic proximity Read ↗

The TRPML3 channel: from gene to function.Semantic proximity Read ↗

The varitint-waddler (Va) deafness mutation in TRPML3 generates constitutive, inward rectifying currents and causes cell degeneration.Semantic proximity Read ↗

TRPML3.Semantic proximity Read ↗

Genetic inactivation of Trpml3 does not lead to hearing and vestibular impairment in mice.Semantic proximity Read ↗

Gain-of-function mutation in TRPML3 causes the mouse Varitint-Waddler phenotype.Semantic proximity Read ↗

The varitint-waddler mouse phenotypes and the TRPML3 ion channel mutation: cause and consequence.Semantic proximity Read ↗

Activating mutation in a mucolipin transient receptor potential channel leads to melanocyte loss in varitint-waddler mice.Semantic proximity Read ↗

Role of TRPML and two-pore channels in endolysosomal cation homeostasis.Semantic proximity Read ↗

Signaling in the Auditory System: Implications in Hair Cell Regeneration and Hearing Function.Semantic proximity Read ↗

On the pathophysiology of DFNA9: Effect of pathogenic variants in the COCH gene on inner ear functioning in human and transgenic mice.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.