Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

In vivo assessment of myocardial glucose uptake by positron emission tomography in adults with the PRKAG2 cardiac syndrome.

Circulation. Cardiovascular imaging - 1 Nov 2009

Ha Andrew C T, Renaud Jennifer M, Dekemp Robert A, Thorn Stephanie, Dasilva Jean, Garrard Linda, Yoshinaga Keiichiro, Abraham Arun, Green Martin S, Beanlands Rob S B, Gollob Michael H

Abstract excerpt

BACKGROUND: The PRKAG2 cardiac syndrome is an inherited metabolic disease of the heart characterized by excessive myocardial glycogen deposition. The biochemical alterations associated with this condition remain controversial and have not previously been studied in affected humans. METHODS AND RESULTS: Positron emission tomography (PET) imaging was used to quantitatively assess myocardial glucose uptake (MGU) in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to AMP-Activated Protein Kinases
Share to Echocardiography

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fluorodeoxyglucose F18

Share to Heart Diseases

Share to Metabolic Diseases

Share to Myocardium

Share to Statistics, Nonparametric

Share to Tomography, Emission-Computed

Source

Identifiers

PMID: 19920047
DOI: 10.1161/CIRCIMAGING.109.853291

Genes

PRKAG2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

In vivo assessment of myocardial glucose uptake by positron emission tomography in adults with the PRKAG2 cardiac syndrome.PMID 19920047Read ↗

Cardiac manifestations of PRKAG2 mutation.Semantic proximity Read ↗

PRKAG2 cardiac syndrome: familial ventricular preexcitation, conduction system disease, and cardiac hypertrophy.Semantic proximity Read ↗

High prevalence of arrhythmic and myocardial complications in patients with cardiac glycogenosis due to PRKAG2 mutations.Semantic proximity Read ↗

Clinical Features and Natural History of PRKAG2 Variant Cardiac Glycogenosis.Semantic proximity Read ↗

Identification of a novel de novo mutation associated with PRKAG2 cardiac syndrome and early onset of heart failure.Semantic proximity Read ↗

Myocardial glucose uptake in patients with the m.3243A > G mutation in mitochondrial DNA.Semantic proximity Read ↗

Cardiovascular magnetic resonance findings in patients with PRKAG2 gene mutations.Semantic proximity Read ↗

Glycogen storage disease as a unifying mechanism of disease in the PRKAG2 cardiac syndrome.Semantic proximity Read ↗

Identification, clinical manifestation and structural mechanisms of mutations in AMPK associated cardiac glycogen storage disease.Semantic proximity Read ↗

Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy.Semantic proximity Read ↗

A PRKAG2 mutation causes biphasic changes in myocardial AMPK activity and does not protect against ischemia.Semantic proximity Read ↗

Novel PRKAG2 mutation responsible for the genetic syndrome of ventricular preexcitation and conduction system disease with childhood onset and absence of cardiac hypertrophy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.