Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The YAP1 homolog-mediated oxidative stress tolerance is crucial for pathogenicity of the necrotrophic fungus Alternaria alternata in citrus.

Molecular plant-microbe interactions : MPMI - 1 Aug 2009

Lin Ching-Hsuan, Yang Siwy Ling, Chung Kuang-Ren

Abstract excerpt

Citrus brown spot disease is caused by the necrotrophic fungus Alternaria alternata. Its pathogenic capability has been thought to depend exclusively on the production of host-selective ACT toxin. However, circumvention of plant defenses is also likely to be important for the disease process. To investigate the fungal response to host-generated reactive oxygen species (ROS), we cloned and characterized the AaAP1...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alternaria
Share to Citrus
Share to Down-Regulation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fungal Proteins

Share to NADPH Oxidases

Share to Oxidative Stress

Share to Reactive Oxygen Species

Share to Virulence

Source

Identifiers

PMID: 19589070
DOI: 10.1094/MPMI-22-8-0942

Genes

YAP1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The YAP1 homolog-mediated oxidative stress tolerance is crucial for pathogenicity of the necrotrophic fungus Alternaria alternata in citrus.PMID 19589070Read ↗

Genomic and transcriptomic analyses of the tangerine pathotype of Alternaria alternata in response to oxidative stress.Semantic proximity Read ↗

How the Pathogenic Fungus Alternaria alternata Copes with Stress via the Response Regulators SSK1 and SHO1.Semantic proximity Read ↗

Contribution of peroxisomes to secondary metabolism and pathogenicity in the fungal plant pathogen Alternaria alternata.Semantic proximity Read ↗

The bZIP transcription factor MoAP1 mediates the oxidative stress response and is critical for pathogenicity of the rice blast fungus Magnaporthe oryzae.Semantic proximity Read ↗

The NADPH Oxidase A of <i>Verticillium dahliae</i> is Essential for Pathogenicity, Normal Development, and Stress Tolerance, and it Interacts with Yap1 to Regulate Redox HomeostasisSemantic proximity Read ↗

A nonribosomal peptide synthetase mediates siderophore production and virulence in the citrus fungal pathogen Alternaria alternata.Semantic proximity Read ↗

The basal transcription factor II H subunit Tfb5 is required for stress response and pathogenicity in the tangerine pathotype of Alternaria alternata.Semantic proximity Read ↗

Aaprb1, a subtilsin-like protease, required for autophagy and virulence of the tangerine pathotype of Alternaria alternata.Semantic proximity Read ↗

An ATP-dependent protease homolog ensures basic standards of survival and pathogenicity for Magnaporthe oryzaeSemantic proximity Read ↗

Characterization of an AP-1-like transcription factor that mediates an oxidative stress response in Kluyveromyces lactis.Semantic proximity Read ↗

A cAMP-Dependent Protein Kinase Gene, aapk1, Is Required for Mycelia Growth, Toxicity and Pathogenicity of Alternaria alternata on TobaccoSemantic proximity Read ↗

Pathogen-induced, NADPH oxidase-derived reactive oxygen intermediates suppress spread of cell death in Arabidopsis thaliana.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.