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Article

A changing gastric environment leads to adaptation of lipopolysaccharide variants in Helicobacter pylori populations during colonization.

PloS one - 11 Jun 2009

Skoglund Anna, Bäckhed Helene Kling, Nilsson Christina, Björkholm Britta, Normark Staffan, Engstrand Lars

Abstract excerpt

The human gastric pathogen Helicobacter pylori colonizes the stomachs of half of the human population, and causes development of peptic ulcer disease and gastric adenocarcinoma. H. pylori-associated chronic atrophic gastritis (ChAG) with loss of the acid-producing parietal cells, is correlated with an increased risk for development of gastric adenocarcinoma. The majority of H. pylori isolates produce...

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Identifiers

PMID: 19517017
DOI: 10.1371/journal.pone.0005885
PMCID: PMC2690825

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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