Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Increased uptake and improved intracellular survival of a teicoplanin-resistant mutant of methicillin-resistant Staphylococcus aureus in non-professional phagocytes.

Chemotherapy - 1 Jan 2009

Renzoni Adriana, Huggler Elzbieta, Kelley William L, Lew Daniel, Vaudaux Pierre

Abstract excerpt

BACKGROUND/AIMS: Endogenous development of glycopeptide-intermediate resistance is linked to multiple genetic and phenotypic changes in clinical and laboratory isolates of Staphylococcus aureus. This study evaluated endocytic uptake and intracellular survival of a teicoplanin-resistant derivative of S. aureus in a human epithelial cell line, and compared these to the isogenic teicoplanin-susceptible parent or a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anti-Bacterial Agents
Share to Cell Line

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Drug Resistance, Bacterial

Share to Methicillin-Resistant Staphylococcus aureus

Share to Microbial Sensitivity Tests

Share to Phagocytes

Share to Teicoplanin

Share to Time Factors

Source

Identifiers

PMID: 19407446
DOI: 10.1159/000215304

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Increased uptake and improved intracellular survival of a teicoplanin-resistant mutant of methicillin-resistant Staphylococcus aureus in non-professional phagocytes.PMID 19407446Read ↗

Staphylococcus aureus Exploits a Non-ribosomal Cyclic Dipeptide to Modulate Survival within Epithelial Cells and PhagocytesSemantic proximity Read ↗

Understanding Staphylococcus aureus internalisation and induction of antimicrobial toleranceSemantic proximity Read ↗

Intracellular staphylococcus aureus: Live-in and let dieSemantic proximity Read ↗

In vivo survival of teicoplanin-resistant Staphylococcus aureus and fitness cost of teicoplanin resistance.Semantic proximity Read ↗

Microscopy-based phenotypic profiling of infection by Staphylococcus aureus clinical isolates reveals intracellular lifestyle as a prevalent featureSemantic proximity Read ↗

Strain-Specific Association of Cytotoxic Activity and Virulence of ClinicalStaphylococcus aureusIsolatesSemantic proximity Read ↗

Intracellular activity of antibiotics in a model of human THP-1 macrophages infected by a Staphylococcus aureus small-colony variant strain isolated from a cystic fibrosis patient: pharmacodynamic evaluation and comparison with isogenic normal-phenotype and revertant strains.Semantic proximity Read ↗

<i>Staphylococcus aureus</i> Escapes More Efficiently from the Phagosome of a Cystic Fibrosis Bronchial Epithelial Cell Line than from Its Normal CounterpartSemantic proximity Read ↗

Intracellular Survival of Staphylococcus aureus in Endothelial Cells: A Matter of Growth or PersistenceSemantic proximity Read ↗

The expression of alpha-haemolysin is required for<i>Staphylococcus aureus</i>phagosomal escape after internalization in CFT-1 cellsSemantic proximity Read ↗

Influence of Sae-regulated and Agr-regulated factors on the escape of<i>Staphylococcus aureus</i>from human macrophagesSemantic proximity Read ↗

The isoleucyl-tRNA synthetase mutation V588F conferring mupirocin resistance in glycopeptide-intermediate Staphylococcus aureus is not associated with a significant fitness burdenSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.