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Article

A point mutation in KINDLIN3 ablates activation of three integrin subfamilies in humans

22 Feb 2009

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 19234460
DOI: 10.1038/nm.1917

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A point mutation in KINDLIN3 ablates activation of three integrin subfamilies in humansPMID 19234460Read ↗

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Biophysical analysis of Kindlin-3 reveals an elongated conformation and maps integrin binding to the membrane-distal β-subunit NPXY motif.Semantic proximity Read ↗

Two mutations in the KINDLIN3 gene of a new leukocyte adhesion deficiency III patient reveal distinct effects on leukocyte function in vitroSemantic proximity Read ↗

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