Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Development of glycopeptide-intermediate resistance by Staphylococcus aureus leads to attenuated infectivity in a rat model of endocarditis.

Microbial pathogenesis - 1 Jan 2000

Majcherczyk Paul A, Barblan Jean-Luc, Moreillon Philippe, Entenza José M

Abstract excerpt

Glycopeptide-intermediate resistant Staphylococcus aureus (GISA) are characterized by multiple changes in the cell wall and an altered expression of global virulence regulators. We investigated whether GISA are affected in their infectivity in a rat model of experimental endocarditis. The glycope...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anti-Bacterial Agents
Share to Bacterial Adhesion
Share to Drug Resistance, Bacterial

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Endocarditis, Bacterial

Share to Glycopeptides

Share to Staphylococcal Infections

Share to Staphylococcus aureus

Share to Virulence

Source

Identifiers

PMID: 18930804
DOI: 10.1016/j.micpath.2008.09.003

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-18930804-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Development of glycopeptide-intermediate resistance by Staphylococcus aureus leads to attenuated infectivity in a rat model of endocarditis.PMID 18930804Read ↗

Comparative mortality of dominant Staphylococcus aureus lineages in human bacteremia and animal infection models.Semantic proximity Read ↗

All Staphylococcus aureus bacteraemia-inducing strains can cause infective endocarditis: Results of GWAS and experimental animal studiesSemantic proximity Read ↗

A comparison of virulence patterns and in vivo fitness between hospital- and community-acquired methicillin-resistant Staphylococcus aureus related to the USA400 clone.Semantic proximity Read ↗

Temperate Prophages Increase Bacterial Adhesin Expression and Virulence in an Experimental Model of Endocarditis Due to Staphylococcus aureus From the CC398 LineageSemantic proximity Read ↗

Vegetation formation in <i>Staphylococcus aureus</i> endocarditis inversely correlates with <i>RNAIII</i> and <i>sarA</i> expression in invasive clonal complex 5 (CC5) isolatesSemantic proximity Read ↗

Phenotypic resistance to thrombin-induced platelet microbicidal protein in vitro is correlated with enhanced virulence in experimental endocarditis due to Staphylococcus aureus.Semantic proximity Read ↗

Vegetation Formation in Staphylococcus Aureus Endocarditis Inversely Correlates With RNAIII and sarA Expression in Invasive Clonal Complex 5 IsolatesSemantic proximity Read ↗

In vivo survival of teicoplanin-resistant Staphylococcus aureus and fitness cost of teicoplanin resistance.Semantic proximity Read ↗

All <i>Staphylococcus aureus</i> bacteraemia strains have the potential to cause infective endocarditis: results of GWAS and experimental animal studiesSemantic proximity Read ↗

Collaborative Cross mice have diverse phenotypic responses to infection with Methicillin-resistant <i>Staphylococcus aureus</i> USA300Semantic proximity Read ↗

Identification of a distinctive phenotype for endocarditis-associated clonal complex 22 MRSA isolates with reduced vancomycin susceptibility.Semantic proximity Read ↗

Association of Novel Streptococcus sanguinis Virulence Factors With Pathogenesis in a Native Valve Infective Endocarditis ModelSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.