Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Dynamics of Neisseria gonorrhoeae attachment: microcolony development, cortical plaque formation, and cytoprotection.

Infection and immunity - 1 Oct 2007

Higashi Dustin L, Lee Shaun W, Snyder Aurelie, Weyand Nathan J, Bakke Antony, So Magdalene

Abstract excerpt

Neisseria gonorrhoeae is the bacterium that causes gonorrhea, a major sexually transmitted disease and a significant cofactor for human immunodeficiency virus transmission. The retactile N. gonorrhoeae type IV pilus (Tfp) mediates twitching motility and attachment. Using live-cell microscopy, we...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenosine Triphosphatases
Share to Bacterial Adhesion
Share to Cytoprotection

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fimbriae, Bacterial

Share to Locomotion

Share to Microscopy

Share to Molecular Motor Proteins

Share to Neisseria gonorrhoeae

Source

Open in PubMed Open full text

Identifiers

PMID: 17682045
DOI: 10.1128/IAI.00687-07
PMCID: PMC2044525

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17682045-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Dynamics of Neisseria gonorrhoeae attachment: microcolony development, cortical plaque formation, and cytoprotection.PMID 17682045Read ↗

Characterization of motility and piliation in pathogenic Neisseria.Semantic proximity Read ↗

The meningococcal PilT protein is required for induction of intimate attachment to epithelial cells following pilus-mediated adhesion.Semantic proximity Read ↗

Discovery of a New Neisseria gonorrhoeae Type IV Pilus Assembly Factor, TfpC.Semantic proximity Read ↗

The actin-based machinery ofTrichomonas vaginalismediates flagellate-amoeboid transition and migration across host tissueSemantic proximity Read ↗

Discovery of a new <i>Neisseria gonorrhoeae</i> Type IV pilus assembly factor, TfpCSemantic proximity Read ↗

Antigenic variation impacts gonococcal lifestyle and antibiotic tolerance by modulating interbacterial forcesSemantic proximity Read ↗

Type IV pilus retraction in pathogenic Neisseria is regulated by the PilC proteinsSemantic proximity Read ↗

Modification of type IV pilus-associated epithelial cell adherence and multicellular behavior by the PilU protein of Neisseria gonorrhoeae.Semantic proximity Read ↗

Pathogenicity and virulence of Chlamydia trachomatis : Insights into host interactions, immune evasion, and intracellular survivalSemantic proximity Read ↗

Characterization of <i>Neisseria gonorrhoeae</i> colonization of macrophages under distinct polarization states and nutrients environmentSemantic proximity Read ↗

Expression of a Clostridium perfringens Type IV Pilin by Neisseria gonorrhoeae Mediates Adherence to Muscle CellsSemantic proximity Read ↗

Extracellular Bacterial Pathogen Induces Host Cell Surface Reorganization to Resist Shear StressSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.