Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Epstein-Barr Virus Infection of Langerhans Cell Precursors as a Mechanism of Oral Epithelial Entry, Persistence, and Reactivation

21 Mar 2007

Abstract excerpt

Epstein-Barr virus (EBV) is a ubiquitous human herpesvirus associated with many malignant and nonmalignant human diseases. Life-long latent EBV persistence occurs in blood-borne B lymphocytes, while EBV intermittently productively replicates in mucosal epithelia. Although several models have previously been proposed, the mechanism of EBV transition between these two reservoirs of infection has not been...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytomegalovirus and herpesvirus research
Share to Histiocytic Disorders and Treatments
Share to Viral-associated cancers and disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 17376908
DOI: 10.1128/jvi.02754-06

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Epstein-Barr Virus Infection of Langerhans Cell Precursors as a Mechanism of Oral Epithelial Entry, Persistence, and ReactivationPMID 17376908Read ↗

Epstein–Barr Virus History and PathogenesisSemantic proximity Read ↗

Modes of infection and oncogenesis by the Epstein–Barr virusSemantic proximity Read ↗

Epstein–Barr virus in the pathogenesis of oral cancersSemantic proximity Read ↗

Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesisSemantic proximity Read ↗

Biology and disease associations of Epstein–Barr virusSemantic proximity Read ↗

Role of Cytotoxic T Lymphocytes in Epstein-Barr Virus-Associated DiseasesSemantic proximity Read ↗

Molecular Basis of Epstein–Barr Virus Latency Establishment and Lytic ReactivationSemantic proximity Read ↗

Epstein–Barr virus latent genesSemantic proximity Read ↗

EBV stimulates TLR‐ and autophagy‐dependent pathways and impairs maturation in plasmacytoid dendritic cells: Implications for viral immune escapeSemantic proximity Read ↗

EBV Association with Lymphomas and Carcinomas in the Oral CompartmentSemantic proximity Read ↗

Epstein–Barr Virus: How Its Lytic Phase Contributes to OncogenesisSemantic proximity Read ↗

Epstein-Barr Virus Infection Promotes Epithelial Cell Growth by Attenuating Differentiation-Dependent Exit from the Cell CycleSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.