Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic interactions of TGA transcription factors in the regulation of pathogenesis-related genes and disease resistance in Arabidopsis.

Plant physiology - 1 May 2007

Kesarwani Meenu, Yoo Jungmin, Dong Xinnian

Abstract excerpt

TGA transcription factors are implicated as regulators of pathogenesis-related (PR) genes because of their physical interaction with the known positive regulator, nonexpresser of PR gene1 (NPR1). A triple-knockout mutant tga2-1 tga5-1 tga6-1 was shown previously to be defective in the induction of PR genes and systemic acquired resistance, confirming their role in disease resistance. However, the contributions of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arabidopsis
Share to Arabidopsis Proteins
Share to Gene Expression Regulation, Plant

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Immunity, Innate

Share to Mutagenesis, Insertional

Share to Nuclear Proteins

Share to Plant Diseases

Share to Plant Proteins

Source

Open in PubMed Open full text

Identifiers

PMID: 17369431
DOI: 10.1104/pp.106.095299
PMCID: PMC1913812

Genes

NPR1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic interactions of TGA transcription factors in the regulation of pathogenesis-related genes and disease resistance in Arabidopsis.PMID 17369431Read ↗

Knockout analysis of Arabidopsis transcription factors TGA2, TGA5, and TGA6 reveals their redundant and essential roles in systemic acquired resistance.Semantic proximity Read ↗

Negative regulation of defense responses in Arabidopsis by two NPR1 paralogs.Semantic proximity Read ↗

Over-expression of TGA5, which encodes a bZIP transcription factor that interacts with NIM1/NPR1, confers SAR-independent resistance in Arabidopsis thaliana to Peronospora parasitica.Semantic proximity Read ↗

In vivo biochemical characterization of transcription factors regulating plant defense response to diseaseSemantic proximity Read ↗

Arabidopsis clade I TGA factors regulate apoplastic defences against the bacterial pathogen Pseudomonas syringae through endoplasmic reticulum-based processes.Semantic proximity Read ↗

Alteration of TGA factor activity in rice results in enhanced tolerance to Xanthomonas oryzae pv. oryzae.Semantic proximity Read ↗

Tobacco transcription factor TGA2.2 is the main component of as-1-binding factor ASF-1 and is involved in salicylic acid- and auxin-inducible expression of as-1-containing target promoters.Semantic proximity Read ↗

In vivo interaction between NPR1 and transcription factor TGA2 leads to salicylic acid-mediated gene activation in Arabidopsis.Semantic proximity Read ↗

An as-1-like motif controls the level of expression of the gene for the pathogenesis-related protein 1a from tobacco.Semantic proximity Read ↗

The TGA Transcription Factors from Clade II Negatively Regulate the Salicylic Acid Accumulation in Arabidopsis.Semantic proximity Read ↗

The Arabidopsis NPR1 disease resistance protein is a novel cofactor that confers redox regulation of DNA binding activity to the basic domain/leucine zipper transcription factor TGA1.Semantic proximity Read ↗

Recessive mutation identifies auxin-repressed protein ARP1, which regulates growth and disease resistance in tobacco.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.