Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration?The beta-N-methylamino-l-alanine (BMAA) paradigm

15 Jan 2007

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alzheimer's disease research and treatments
Share to Amyotrophic Lateral Sclerosis Research
Share to Neurological diseases and metabolism

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 17296249
DOI: 10.1016/j.neuint.2006.12.011

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Is there a role for naturally occurring cyanobacterial toxins in neurodegeneration?The beta-N-methylamino-l-alanine (BMAA) paradigmPMID 17296249Read ↗

A critical review of the postulated role of the non-essential amino acid, β-N-methylamino-L-alanine, in neurodegenerative disease in humansSemantic proximity Read ↗

Cellular and Molecular Aspects of the β-N-Methylamino-l-alanine (BMAA) Mode of Action within the Neurodegenerative Pathway: Facts and ControversySemantic proximity Read ↗

The Proposed Neurotoxin β-N-Methylamino-l-Alanine (BMAA) Is Taken up through Amino-Acid Transport Systems in the Cyanobacterium Anabaena PCC 7120.Semantic proximity Read ↗

Evaluation of cyanotoxin L-BMAA effect on α-synuclein and TDP43 proteinopathySemantic proximity Read ↗

Occurrence of β-N-methylamino-l-alanine (BMAA) and Isomers in Aquatic Environments and Aquatic Food Sources for HumansSemantic proximity Read ↗

Neuropathological Mechanisms of β-N-Methylamino-L-Alanine (BMAA) with a Focus on Iron Overload and FerroptosisSemantic proximity Read ↗

Cyanobacteria, Cyanotoxins, and Neurodegenerative Diseases: Dangerous Liaisons.Semantic proximity Read ↗

The cyanotoxin 2,4-DAB enhances mortality and causes behavioral and molecular dysfunctions associated with neurodegeneration in larval zebrafishSemantic proximity Read ↗

β-N-methylamino-l-alanine causes neurological and pathological phenotypes mimicking Amyotrophic Lateral Sclerosis (ALS): the first step towards an experimental model for sporadic ALS.Semantic proximity Read ↗

The Cyanotoxin 2,4-DAB Reduces Viability and Causes Behavioral and Molecular Dysfunctions Associated with Neurodegeneration in Larval ZebrafishSemantic proximity Read ↗

Biosynthesis of the proteins containing neurotoxin <i>β</i> - <i>N</i> -methylamino-L-alanine in marine diatomsSemantic proximity Read ↗

d-Alanine: Distribution, origin, physiological relevance, and implications in diseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.