Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Characterization of dopaminergic dysfunction in familial progressive supranuclear palsy: an 18F-dopa PET study.

Journal of neural transmission (Vienna, Austria : 1996) - 1 Mar 2007

Tai Y F, Ahsan R L, de Yébenes J G, Pavese N, Brooks D J, Piccini P

Abstract excerpt

We analyzed (18)F-dopa PET data from 11 members of kindreds with familial progressive supranuclear palsy (PSP) to characterize their cerebral dopaminergic dysfunction. Three clinically-affected PSP patients showed reduced (18)F-dopa uptake in the striatum, orbitofrontal cortex and amygdala. One a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Amygdala
Share to Corpus Striatum
Share to Dihydroxyphenylalanine

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Dopamine

Share to Limbic System

Share to Neurocognitive Disorders

Share to Positron-Emission Tomography

Share to Supranuclear Palsy, Progressive

Source

Identifiers

PMID: 16897607
DOI: 10.1007/s00702-006-0536-0

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-16897607-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Characterization of dopaminergic dysfunction in familial progressive supranuclear palsy: an 18F-dopa PET study.PMID 16897607Read ↗

Familial progressive supranuclear palsy: detection of subclinical cases using 18F-dopa and 18fluorodeoxyglucose positron emission tomography.Semantic proximity Read ↗

Progressive supranuclear palsy: a systematic review.Semantic proximity Read ↗

Dopaminergic function in familial Parkinson's disease: A clinical and <sup>18</sup>F‐dopa positron emission tomography studySemantic proximity Read ↗

The diagnosis of progressive supranuclear palsy: current opinions and challenges.Semantic proximity Read ↗

Progressive Supranuclear Palsy: an Update.Semantic proximity Read ↗

Spinocerebellar ataxia type 2 presenting as familial levodopa‐responsive parkinsonismSemantic proximity Read ↗

Progressive supranuclear palsy: current approach and challenges to diagnosis and treatment.Semantic proximity Read ↗

Neuropathology of variants of progressive supranuclear palsy.Semantic proximity Read ↗

Clinical, cognitive, and morphometric profiles of progressive supranuclear palsy phenotypes.Semantic proximity Read ↗

Familial striatal degeneration: New mutation and neuroimaging clues.Semantic proximity Read ↗

The phenotypic spectrum of progressive supranuclear palsy.Semantic proximity Read ↗

Two large British kindreds with familial Parkinson's disease: a clinico-pathological and genetic study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.