Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

High-Resolution Genomic and Expression Profiling Reveals 105 Putative Amplification Target Genes in Pancreatic Cancer

1 Sept 2004

Abstract excerpt

Comparative genomic hybridization (CGH) studies have provided a wealth of information on common copy number aberrations in pancreatic cancer, but the genes affected by these aberrations are largely unknown. To identify putative amplification target genes in pancreatic cancer, we performed a parallel copy number and expression survey in 13 pancreatic cancer cell lines using a 12,232-clone cDNA microarray,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Genomics and Diagnostics
Share to Genomic variations and chromosomal abnormalities
Share to Pancreatic and Hepatic Oncology Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 15548351
DOI: 10.1593/neo.04130

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

High-Resolution Genomic and Expression Profiling Reveals 105 Putative Amplification Target Genes in Pancreatic CancerPMID 15548351Read ↗

Genome-Wide Array-Based Comparative Genomic Hybridization Reveals Multiple Amplification Targets and Novel Homozygous Deletions in Pancreatic Carcinoma Cell LinesSemantic proximity Read ↗

High-resolution characterization of the pancreatic adenocarcinoma genomeSemantic proximity Read ↗

Characterization of the 7q21‐q22 amplicon identifies ARPC1A, a subunit of the Arp2/3 complex, as a regulator of cell migration and invasion in pancreatic cancerSemantic proximity Read ↗

Array-Based Comparative Genomic Hybridization Identifies Localized DNA Amplifications and Homozygous Deletions in Pancreatic CancerSemantic proximity Read ↗

Integrated analysis of copy number alterations and loss of heterozygosity in human pancreatic cancer using a high-resolution, single nucleotide polymorphism array.Semantic proximity Read ↗

Genome‐wide array‐based comparative genomic hybridization analysis of pancreatic adenocarcinoma: Identification of genetic indicators that predict patient outcomeSemantic proximity Read ↗

Microarray analyses reveal strong influence of DNA copy number alterations on the transcriptional patterns in pancreatic cancer: implications for the interpretation of genomic amplificationsSemantic proximity Read ↗

Pancreatic adenocarcinoma—Genetic portrait from chromosomes to microarraysSemantic proximity Read ↗

Expression analysis of pancreatic cancer cell lines reveals association of enhanced gene transcription and genomic amplifications at the 8q22.1 and 8q24.22 loci.Semantic proximity Read ↗

Copy number alterations in pancreatic cancer identify recurrent PAK4 amplification.Semantic proximity Read ↗

Analysis of genomic DNA alterations and mRNA expression patterns in a panel of human pancreatic cancer cell linesSemantic proximity Read ↗

Genome-wide DNA copy number analysis in pancreatic cancer using high-density single nucleotide polymorphism arrays.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.