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Article

Meiotic catastrophe and retrotransposon reactivation in male germ cells lacking Dnmt3L

18 Aug 2004

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Share to Epigenetics and DNA Methylation
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Share to Prenatal Screening and Diagnostics

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15318244
DOI: 10.1038/nature02886

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Meiotic catastrophe and retrotransposon reactivation in male germ cells lacking Dnmt3LPMID 15318244Read ↗

Meiotic and epigenetic aberrations in Dnmt3L-deficient male germ cells.Semantic proximity Read ↗

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Origins and consequences of DNA damage in male germ cellsSemantic proximity Read ↗

Meiotic Errors Activate Checkpoints that Improve Gamete Quality without Triggering Apoptosis in Male Germ CellsSemantic proximity Read ↗

DNMT3A-dependent DNA methylation is required for spermatogonial stem cells to commit to spermatogenesisSemantic proximity Read ↗

Transcriptional CheckpointsDetermining the Fate of Male Germ CellsSemantic proximity Read ↗

Lsh is required for meiotic chromosome synapsis and retrotransposon silencing in female germ cellsSemantic proximity Read ↗

How do male germ cells handle DNA damage?Semantic proximity Read ↗

The mitotic STAG3–cohesin complex shapes male germline nucleomeSemantic proximity Read ↗

Genetics of mammalian meiosis: regulation, dynamics and impact on fertilitySemantic proximity Read ↗

Haploinsufficiency of the paternal-effect gene Dnmt3L results in transient DNA hypomethylation in progenitor cells of the male germline.Semantic proximity Read ↗

MCM9 deficiency delays primordial germ cell proliferation independent of the ATM pathway.Semantic proximity Read ↗

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