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Article

Intramuscular gene transfer of interleukin-10 cDNA reduces atherosclerosis in apolipoprotein E-knockout mice

17 Nov 2003

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Share to Adipokines, Inflammation, and Metabolic Diseases
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Identifiers

PMID: 14709353
DOI: 10.1016/j.atherosclerosis.2003.08.032

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Related research

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Intramuscular gene transfer of interleukin-10 cDNA reduces atherosclerosis in apolipoprotein E-knockout micePMID 14709353Read ↗

Interleukin-10 deficiency increases atherosclerosis, thrombosis, and low-density lipoproteins in apolipoprotein E knockout mice.Semantic proximity Read ↗

Overexpression of interleukin-10 by activated T lymphocytes inhibits atherosclerosis in LDL receptor-deficient Mice by altering lymphocyte and macrophage phenotypes.Semantic proximity Read ↗

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New Anti–Monocyte Chemoattractant Protein-1 Gene Therapy Attenuates Atherosclerosis in Apolipoprotein E–Knockout MiceSemantic proximity Read ↗

Enhanced expression of matrix metalloproteinase-3, -12, and -13 mRNAs in the aortas of apolipoprotein E-deficient mice with advanced atherosclerosis.Semantic proximity Read ↗

Apolipoprotein E and Atherosclerosis: From Lipoprotein Metabolism to MicroRNA Control of InflammationSemantic proximity Read ↗

The anti-atherosclerosis effect of anakinra, a recombinant human interleukin-1 receptor antagonist, in apolipoprotein E knockout miceSemantic proximity Read ↗

Endothelial nitric oxide synthase gene transfer restores endothelium-dependent relaxations and attenuates lesion formation in carotid arteries in apolipoprotein E-deficient mice.Semantic proximity Read ↗

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