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Article

BAK Alters Neuronal Excitability and Can Switch from Anti- to Pro-Death Function during Postnatal Development

1 Apr 2003

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12689595
DOI: 10.1016/s1534-5807(03)00091-1

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BAK Alters Neuronal Excitability and Can Switch from Anti- to Pro-Death Function during Postnatal DevelopmentPMID 12689595Read ↗

Developmental Attenuation of Neuronal Apoptosis by Neural-Specific Splicing of Bak1 Microexon.Semantic proximity Read ↗

Neuron-specific Bcl-2 homology 3 domain-only splice variant of Bak is anti-apoptotic in neurons, but pro-apoptotic in non-neuronal cells.Semantic proximity Read ↗

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Targeted Inactivation of Bax Reveals a Subtype-Specific Mechanism of Cajal-Retzius Neuron Death in the Postnatal Cerebral CortexSemantic proximity Read ↗

Dynamics of expression of apoptosis-regulatory proteins Bid, Bcl-2, Bcl-X, Bax and Bak during development of murine nervous systemSemantic proximity Read ↗

Chronic PERK induction promotes Alzheimer-like neuropathology in Down syndrome: Insights for therapeutic interventionSemantic proximity Read ↗

K+ Channels in ApoptosisSemantic proximity Read ↗

More alive than dead: non-apoptotic roles for caspases in neuronal development, plasticity and diseaseSemantic proximity Read ↗

The VDAC2-BAK rheostat controls thymocyte survival.Semantic proximity Read ↗

Before epilepsy unfolds: opening up the potassium door in neonatal seizures.Semantic proximity Read ↗

The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues.Semantic proximity Read ↗

Electrophysiological behavior of neonatal astrocytes in hippocampal stratum radiatum.Semantic proximity Read ↗

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