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Article

Cell surface-binding motifs of L2 that facilitate papillomavirus infection.

Journal of virology - 1 Mar 2003

Yang Rongcun, Day Patricia M, Yutzy William H, Lin Ken-Yu, Hung Chien-Fu, Roden Richard B S

Abstract excerpt

Human papillomavirus type 16 (HPV16) is the primary etiologic agent of cervical carcinoma, whereas bovine papillomavirus type 1 (BPV1) causes benign fibropapillomas. However, the capsid proteins, L1 and L2, of these divergent papillomaviruses exhibit functional conservation. A peptide comprising residues 1 to 88 of BPV1 L2 binds to a variety of cell lines, but not to the monocyte-derived cell line D32, and blocks...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 12610128
DOI: 10.1128/jvi.77.6.3531-3541.2003
PMCID: PMC149523

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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