Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Heterozygosity for the factor V Leiden (G1691A) mutation predisposes renal transplant recipients to thrombotic complications and graft loss.

Transplantation - 15 Aug 2001

Wüthrich R P, Cicvara-Muzar S, Booy C, Maly F E

Abstract excerpt

BACKGROUND: Heterozygosity for a mutation in the coagulation factor V gene (factor V Leiden; FVL) leads to resistance to activated protein C and represents the most common cause of inherited thrombophilia. FVL is associated with a high risk for thromboembolic events and might be a risk factor for venous thrombosis and early graft loss in renal transplant recipients. METHODS: We studied a cohort of 202 renal...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Factor V
Share to Graft Rejection
Share to Kidney Diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Kidney Transplantation

Share to Pulmonary Embolism

Share to Renal Circulation

Share to Thrombosis

Share to Venous Thrombosis

Source

Identifiers

PMID: 11502996
DOI: 10.1097/00007890-200108150-00037

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Heterozygosity for the factor V Leiden (G1691A) mutation predisposes renal transplant recipients to thrombotic complications and graft loss.PMID 11502996Read ↗

Factor V Leiden mutation: potential thrombogenic role in renal vein, dialysis graft and transplant vascular thrombosis.Semantic proximity Read ↗

Outcome of kidney transplantation in patients with inherited thrombophilia: data of a prospective study.Semantic proximity Read ↗

Factor V R506Q mutation (activated protein C resistance) is an additional risk factor for early renal graft loss associated with acute vascular rejection.Semantic proximity Read ↗

The factor V Leiden (R506Q) mutation and risk of thrombosis in renal transplant recipients.Semantic proximity Read ↗

Factor V Leiden mutation is a risk factor for hepatic artery thrombosis in liver transplantation.Semantic proximity Read ↗

Donor factor V Leiden mutation and vascular thrombosis following liver transplantation.Semantic proximity Read ↗

Association of factor V Leiden mutation with delayed graft function, acute rejection episodes and long-term graft dysfunction in kidney transplant recipients.Semantic proximity Read ↗

Genetic predisposition to thrombosis in renal transplant recipients: the factor V Q506 (Leiden) allele.Semantic proximity Read ↗

Is screening for factor V Leiden and prothrombin G20210A mutations in renal transplantation worthwhile? Results of a large single-center U.K. study.Semantic proximity Read ↗

Factor V Leiden and prothrombin G20210A mutations in Thai patients awaiting kidney transplant.Semantic proximity Read ↗

Thrombophilia and avascular necrosis of femoral head in kidney allograft recipients.Semantic proximity Read ↗

Thromboembolism in heart transplantation: role of prothrombin G20210A and factor V Leiden.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.