Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Repression of GADD153/CHOP by NF-κB: a possible cellular defense against endoplasmic reticulum stress-induced cell death

19 Apr 2001

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related Molecular Pathways
Share to Cell death mechanisms and regulation
Share to Endoplasmic Reticulum Stress and Disease

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 11360202
DOI: 10.1038/sj.onc.1204292

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Repression of GADD153/CHOP by NF-κB: a possible cellular defense against endoplasmic reticulum stress-induced cell deathPMID 11360202Read ↗

Stress-Inducible Transcription Factor CHOP/gadd153 Induces Apoptosis in Mammalian Cells via p38 Kinase-Dependent and -Independent MechanismsSemantic proximity Read ↗

Roles of CHOP/GADD153 in endoplasmic reticulum stressSemantic proximity Read ↗

Transcription Factor C/EBP Homologous Protein in Health and DiseasesSemantic proximity Read ↗

New insights into the roles of CHOP-induced apoptosis in ER stressSemantic proximity Read ↗

The Crosstalk of Endoplasmic Reticulum (ER) Stress Pathways with NF-κB: Complex Mechanisms Relevant for Cancer, Inflammation and InfectionSemantic proximity Read ↗

Regulation of necrotic cell death: p53, PARP1 and cyclophilin D-overlapping pathways of regulated necrosis?Semantic proximity Read ↗

CHOP Potentially Co-Operates with FOXO3a in Neuronal Cells to Regulate PUMA and BIM Expression in Response to ER StressSemantic proximity Read ↗

Crosstalk between ER stress and immunogenic cell deathSemantic proximity Read ↗

Molecular Symbiosis of CHOP and C/EBPβ Isoform LIP Contributes to Endoplasmic Reticulum Stress-Induced ApoptosisSemantic proximity Read ↗

The transcription factor CHOP, a central component of the transcriptional regulatory network induced upon CCl4 intoxication in mouse liver, is not a critical mediator of hepatotoxicitySemantic proximity Read ↗

Differential regulation of CHOP-10/GADD153 gene expression by MAPK signaling in pancreatic β-cellsSemantic proximity Read ↗

Critical and Functional Regulation of CHOP (C/EBP Homologous Protein) through the N-terminal PortionSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.