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Article

GSTM1 null polymorphism and susceptibility to endometriosis and ovarian cancer.

Carcinogenesis - 1 Jan 2001

Baxter S W, Thomas E J, Campbell I G

Abstract excerpt

It is likely that heritable genetic factors contribute to the development of endometriosis, which is a putative precursor of the endometrioid and clear cell histological subtypes of ovarian cancer. The phase II glutathione S-transferases (GSTs) are a family of enzymes responsible for metabolism of a broad range of xenobiotics and carcinogens. Allelic variants of GSTs that have impaired detoxification function may...

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Source

Identifiers

PMID: 11159742
DOI: 10.1093/carcin/22.1.63

Genes

GSTM1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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GSTM1 null polymorphism and susceptibility to endometriosis and ovarian cancer.PMID 11159742Read ↗

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Null genotypes of GSTM1 and GSTT1 and endometriosis risk: a meta-analysis of 25 case-control studies.Semantic proximity Read ↗

Functional genetic polymorphisms and female reproductive disorders: part II--endometriosis.Semantic proximity Read ↗

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Glutathione S-transferase M1 and T1 gene polymorphisms in Brazilian women with endometriosis.Semantic proximity Read ↗

Polymorphisms of glutathione S-transferase M1 (GSTM1) and T1 (GSTT1) and endometriosis risk: a meta-analysis.Semantic proximity Read ↗

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