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Article

Risk of venous thromboembolism associated with the insertion/deletion polymorphism in the angiotensin-converting enzyme gene.

Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis - 1 Jul 2000

González Ordóñez A J, Fernández Carreira J M, Medina Rodríguez J M, Martín Sánchez L, Alvarez Díaz R, Alvarez Martinez M V, Coto Garcia E

Abstract excerpt

The circulating levels of angiotensin I-converting enzyme (ACE) are linked with a 287-base pair insertion/deletion (I/D) polymorphism at intron 16 of the ACE gene. Thus, the homozygous deletion (D/D genotype) could cause chronic vasoconstriction, arterial hypertension and, possibly, coronary arte...

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Share to Gene Deletion
Share to Mutagenesis, Insertional

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Peptidyl-Dipeptidase A

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Share to Pulmonary Embolism

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Source

Identifiers

PMID: 10937809
DOI: 10.1097/00001721-200007000-00011

Genes

ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-10937809-2026-09-02.1. Manifest publication-graph-cohort-2026-09-02.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

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Risk of venous thromboembolism associated with the insertion/deletion polymorphism in the angiotensin-converting enzyme gene.PMID 10937809Read ↗

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