Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genetic interactions between the chlorate-resistant mutant cr 8 8 and the photomorphogenic mutants cop1 and hy5.

The Plant cell - 1 Feb 2000

Cao D, Lin Y, Cheng C L

Abstract excerpt

The chlorate-resistant mutant cr88 is defective in photomorphogenesis, as shown by the phenotypes of long hypocotyls in red light and yellow cotyledons under all light conditions. A subset of light-regulated genes is expressed at subnormal levels in cr88. To analyze further the role that CR88 plays in photomorphogenesis, we investigated the genetic interactions between cr88 and mutants of two other loci affecting...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arabidopsis Proteins
Share to Basic-Leucine Zipper Transcription Factors

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Chlorates

Share to Cotyledon

Share to Photosynthesis

Share to RNA, Plant

Share to Ubiquitin-Protein Ligases

Source

Open in PubMed Open full text

Identifiers

PMID: 10662857
DOI: 10.1105/tpc.12.2.199
PMCID: PMC139758

Genes

COP1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genetic interactions between the chlorate-resistant mutant cr 8 8 and the photomorphogenic mutants cop1 and hy5.PMID 10662857Read ↗

The chlorate-resistant and photomorphogenesis-defective mutant cr88 encodes a chloroplast-targeted HSP90.Semantic proximity Read ↗

Promotion of photomorphogenesis by COP1.Semantic proximity Read ↗

Overexpression of Arabidopsis COP1 results in partial suppression of light-mediated development: evidence for a light-inactivable repressor of photomorphogenesis.Semantic proximity Read ↗

Procuste1 mutants identify two distinct genetic pathways controlling hypocotyl cell elongation, respectively in dark- and light-grown Arabidopsis seedlings.Semantic proximity Read ↗

A missense mutation in WRKY32 converts its function from a positive regulator to a repressor of photomorphogenesis.Semantic proximity Read ↗

HFR1, a phytochrome A-signalling component, acts in a separate pathway from HY5, downstream of COP1 in Arabidopsis thaliana.Semantic proximity Read ↗

Genomic evidence for COP1 as a repressor of light-regulated gene expression and development in Arabidopsis.Semantic proximity Read ↗

Analysis of the mutational effects of the COP/DET/FUS loci on genome expression profiles reveals their overlapping yet not identical roles in regulating Arabidopsis seedling development.Semantic proximity Read ↗

COP9: a new genetic locus involved in light-regulated development and gene expression in arabidopsis.Semantic proximity Read ↗

Genetic and molecular analysis of an allelic series of cop1 mutants suggests functional roles for the multiple protein domains.Semantic proximity Read ↗

Expression of an N-terminal fragment of COP1 confers a dominant-negative effect on light-regulated seedling development in Arabidopsis.Semantic proximity Read ↗

The Photomorphogenic Central Repressor COP1: Conservation and Functional Diversification during Evolution.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.