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Article

The inlAB locus mediates the entry of Listeria monocytogenes into hepatocytes in vivo.

The Journal of experimental medicine - 1 Feb 1996

Gaillard J L, Jaubert F, Berche P

Abstract excerpt

The intracellular parasite Listeria monocytogenes is able to induce its internalization by cultured mammalian cells that are not normally phagocytic. This process requires the expression of the chromosomal locus inlAB. We studied the virulence of an inlAB mutant and of its parent in murine lister...

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PMID: 8627149
DOI: 10.1084/jem.183.2.359
PMCID: PMC2192459

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-8627149-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

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The inlAB locus mediates the entry of Listeria monocytogenes into hepatocytes in vivo.PMID 8627149Read ↗

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InlA- but not InlB-mediated internalization of Listeria monocytogenes by non-phagocytic mammalian cells needs the support of other internalins.Semantic proximity Read ↗

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Naturally occurring InlB variants that support intragastric Listeria monocytogenes infection in mice.Semantic proximity Read ↗

InlP, a New Virulence Factor with Strong Placental TropismSemantic proximity Read ↗

Mutants of Listeria monocytogenes defective in In vitro invasion and cell-to-cell spreading still invade and proliferate in hepatocytes of neutropenic mice.Semantic proximity Read ↗

InlB: an invasion protein of Listeria monocytogenes with a novel type of surface association.Semantic proximity Read ↗

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Lmo0171, a novel internalin-like protein, determines cell morphology of Listeria monocytogenes and its ability to invade human cell lines.Semantic proximity Read ↗

Disruption of InlC2 enhances the internalization of Listeria monocytogenes by epithelial cellsSemantic proximity Read ↗

Internalin B is essential for adhesion and mediates the invasion of Listeria monocytogenes into human endothelial cells.Semantic proximity Read ↗

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