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Article

Anti-Ku alloimmunization due to a KEL02N.19 allele causing Kell-null phenotype despite KEL02 genotype prediction.

Vox sanguinis - 1 May 2026

Dara Ravi C, Madkour Rania, Bensing Kathleen, Alzaabi Eiman, Sadiq Ihsanullah, Chada Nyaradzo, Mawad Ghada H, Puca Kathleen E, Algora Manuel

Abstract excerpt

BACKGROUND AND OBJECTIVES: Alloantibodies against high-prevalence antigens can create significant barriers to transfusion, particularly in patients with complex medical needs. Serological investigations include use of enzyme-treated red cells and dithiothreitol (DTT) testing, which can enhance or diminish antibody reactivity, providing insight into potential antibody specificity. Additionally, molecular testing...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 41692422
DOI: 10.1111/vox.70209

Genes

KEL

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 23 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

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Anti-Ku alloimmunization due to a KEL*02N.19 allele causing Kell-null phenotype despite KEL*02 genotype prediction.PMID 41692422Read ↗

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