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Article

Bisphenol A exposure induces polycystic ovarian syndrome phenotype B and reduces fertility in adolescent female rats

20 Jan 2025

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 40440795
DOI: 10.1016/j.rbmo.2025.104831

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

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Bisphenol A exposure induces polycystic ovarian syndrome phenotype B and reduces fertility in adolescent female ratsPMID 40440795Read ↗

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Bisphenol A exposure modulates reproductive and endocrine system, mitochondrial function and cellular senescence in female adult rats: A hallmarks of polycystic ovarian syndrome phenotype.Semantic proximity Read ↗

The Association Between Bisphenol A and Polycystic Ovarian Syndrome: A Case-Control Study.Semantic proximity Read ↗

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Bisphenol A and bisphenol S both disrupt ovine granulosa cell steroidogenesis but through different molecular pathwaysSemantic proximity Read ↗

Does Bisphenol A Confer Risk of Neurodevelopmental Disorders? What We’ve Learned from Developmental Neurotoxicity Studies in Animal ModelsSemantic proximity Read ↗

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Bisphenol Z exposure inhibits oocyte meiotic maturation by rupturing mitochondrial functionSemantic proximity Read ↗

Serum bisphenol S levels are associated with decreased ovarian reserve function: a single-center studySemantic proximity Read ↗

Bisphenol A exposure modifies DNA methylation of imprint genes in mouse fetal germ cellsSemantic proximity Read ↗

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