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Perinatal loss of galactosylceramidase in both oligodendrocytes and microglia is crucial for the pathogenesis of Krabbe disease in mice

11 May 2024

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PMID: 38734898
DOI: 10.1016/j.ymthe.2024.05.019

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Perinatal loss of galactosylceramidase in both oligodendrocytes and microglia is crucial for the pathogenesis of Krabbe disease in micePMID 38734898Read ↗

Brainstem development requires galactosylceramidase and is critical for the pathogenesis of Krabbe DiseaseSemantic proximity Read ↗

Generation of a LacZ reporter transgenic mouse line for the stereological analysis of oligodendrocyte loss in galactosylceramidase deficiency.Semantic proximity Read ↗

Brainstem development requires galactosylceramidase and is critical for pathogenesis in a model of Krabbe disease.Semantic proximity Read ↗

Galactosylceramidase deficiency and pathological abnormalities in cerebral white matter of Krabbe disease.Semantic proximity Read ↗

Molecular cloning and knockdown of galactocerebrosidase in zebrafish: New insights into the pathogenesis of Krabbe's diseaseSemantic proximity Read ↗

The galactocerebrosidase enzyme contributes to maintain a functional neurogenic niche during early post-natal CNS developmentSemantic proximity Read ↗

Brain accumulation of lactosylceramide characterizes GALC deficiency in a zebrafish model of Krabbe diseaseSemantic proximity Read ↗

Missense mutation in mouse GALC mimics human gene defect and offers new insights into Krabbe disease.Semantic proximity Read ↗

Myelination in the absence of galactolipids and proteolipid proteins.Semantic proximity Read ↗

Insights into the Pathogenesis and Treatment of Krabbe Disease.Semantic proximity Read ↗

Cell-specific deletion of glucosylceramide synthase in brain leads to severe neural defects after birth.Semantic proximity Read ↗

Krabbe disease: New hope for an old diseaseSemantic proximity Read ↗

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