Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Comprehensive Analysis of METTLs (METTL1/13/18/21A/23/25/2A/2B/5/6/9) and Associated mRNA Risk Signature in Hepatocellular Carcinoma.

Analytical cellular pathology (Amsterdam) - 1 Jan 2023

Wang Haoyu, Hu Shangshang, Nie Junjie, Qin Xiaodan, Zhang Xu, Wang Qian, Li John Zhong

Abstract excerpt

Currently, 80%-90% of liver cancers are hepatocellular carcinomas (HCC). HCC patients develop insidiously and have an inferior prognosis. The methyltransferase-like (METTL) family principal members are strongly associated with epigenetic and tumor progression. The present study mainly analyzed the value of METTLs (METTL1/13/18/21A/23/25/2A/2B/5/6/9) and associated mRNA risk signature for HCC. METTLs expression is...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carcinoma, Hepatocellular
Share to Epithelial-Mesenchymal Transition

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Liver Neoplasms

Share to Methyltransferases

Share to Mutation

Share to Neoplastic Stem Cells

Share to Tumor Microenvironment

Source

Open in PubMed Open full text

Identifiers

PMID: 38130905
DOI: 10.1155/2023/6007431
PMCID: PMC10735724

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Comprehensive Analysis of METTLs (METTL1/13/18/21A/23/25/2A/2B/5/6/9) and Associated mRNA Risk Signature in Hepatocellular Carcinoma.PMID 38130905Read ↗

METTL5 serves as a diagnostic and prognostic biomarker in hepatocellular carcinoma by influencing the immune microenvironmentSemantic proximity Read ↗

Identification of an Epithelial-Mesenchymal Transition-Related Long Non-coding RNA Prognostic Signature for Hepatocellular CarcinomaSemantic proximity Read ↗

Development of N6-methyladenosine Related Signature as New Biomarker for Prognosis of Hepatocellular Carcinoma and Correlates with Sorafenib and anti-PD-1 Immunotherapy Treatment ResponseSemantic proximity Read ↗

Construction of a Prognostic m6A-related lncRNA Signature to Predict Immunotherapy and Chemotherapy Response in Hepatocellular CarcinomaSemantic proximity Read ↗

Identification of m6A methyltransferase-related lncRNA signature for predicting immunotherapy and prognosis in patients with hepatocellular carcinomaSemantic proximity Read ↗

DNA methylation-driven genes for constructing diagnostic, prognostic, and recurrence models for hepatocellular carcinomaSemantic proximity Read ↗

Significance of m 6A Methyltransferase‑Related lncRNAs in the Prognosis and Immunotherapy of Hepatocellular CarcinomaSemantic proximity Read ↗

M7G methylated core genes (METTL1 and WDR4) and associated RNA risk signatures are associated with prognosis and immune escape in HCCSemantic proximity Read ↗

Diagnosis and prognosis models for hepatocellular carcinoma patient's management based on tumor mutation burden.Semantic proximity Read ↗

M 6A-related genes characterization in hepatocellular cancer identifies prognostic relevant gene signaturesSemantic proximity Read ↗

Investigation of mitochondrial DNA methylation-related prognostic biomarkers in hepatocellular carcinoma using The Cancer Genome Atlas (TCGA) databaseSemantic proximity Read ↗

Integrated Analysis of Methylation-driven Genes and Pretreatment Prognostic Factors in Patients with Hepatocellular CarcinomaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.