Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Differential transcriptome response following infection of porcine ileal enteroids with species A and C rotaviruses.

Virology journal - 17 Oct 2023

Raev Sergei A, Raque Molly, Kick Maryssa K, Saif Linda J, Vlasova Anastasia N

Abstract excerpt

BACKGROUND: Rotavirus C (RVC) is the major causative agent of acute gastroenteritis in suckling piglets, while most RVAs mostly affect weaned animals. Besides, while most RVA strains can be propagated in MA-104 and other continuous cell lines, attempts to isolate and culture RVC strains remain largely unsuccessful. The host factors associated with these unique RVC characteristics remain unknown. METHODS: In this...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Gastroenteritis
Share to Genotype
Share to Phylogeny

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Rotavirus

Share to Rotavirus Infections

Share to Swine Diseases

Share to Transcriptome

Source

Open in PubMed Open full text

Identifiers

PMID: 37848925
DOI: 10.1186/s12985-023-02207-8
PMCID: PMC10580564

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Differential transcriptome response following infection of porcine ileal enteroids with species A and C rotaviruses.PMID 37848925Read ↗

Host Cell Response to Rotavirus Infection with Emphasis on Virus-Glycan Interactions, Cholesterol Metabolism, and Innate Immunity.Semantic proximity Read ↗

Swine Enteric Coronaviruses (PEDV, TGEV, and PDCoV) Induce Divergent Interferon-Stimulated Gene Responses and Antigen Presentation in Porcine Intestinal EnteroidsSemantic proximity Read ↗

Porcine Intestinal Enteroids: a New Model for Studying Enteric Coronavirus Porcine Epidemic Diarrhea Virus Infection and the Host Innate ResponseSemantic proximity Read ↗

Senecavirus a can replicate in apical-out porcine intestinal organoids and induce stress granules and innate immune responseSemantic proximity Read ↗

Rotavirus C Replication in Porcine Intestinal Enteroids Reveals Roles for Cellular Cholesterol and Sialic AcidsSemantic proximity Read ↗

Infection of porcine small intestinal enteroids with human and pig rotavirus A strains reveals contrasting roles for histo-blood group antigens and terminal sialic acidsSemantic proximity Read ↗

Porcine epidemic diarrhea virus infection promotes Peyer’s patch immune induction and epithelial defense via single-cell transcriptional reprogrammingSemantic proximity Read ↗

Pathogenicity of porcine G9P[23] and G9P[7] rotaviruses in piglets.Semantic proximity Read ↗

The role of innate immune responses against two strains of PEDV (S INDEL and non-S INDEL) in newborn and weaned piglets inoculated by combined orogastric and intranasal routesSemantic proximity Read ↗

The dynamics of a Chinese porcine G9P[23] rotavirus production in MA-104 cells and intestines of 3-day-old pigletsSemantic proximity Read ↗

PEDV and PDCoV Pathogenesis: The Interplay Between Host Innate Immune Responses and Porcine Enteric CoronavirusesSemantic proximity Read ↗

Aminopeptidase N Expression, Not Interferon Responses, Determines the Intestinal Segmental Tropism of Porcine DeltacoronavirusSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.