Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Autism Spectrum Disorder: A Neuro-Immunometabolic Hypothesis of the Developmental Origins

26 Jun 2023

Abstract excerpt

Fetal neuroinflammation and prenatal stress (PS) may contribute to lifelong neurological disabilities. Astrocytes and microglia, among the brain's non-neuronal "glia" cell populations, play a pivotal role in neurodevelopment and predisposition to and initiation of disease throughout lifespan. One of the most common neurodevelopmental disorders manifesting between 1-4 years of age is the autism spectrum disorder...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autism Spectrum Disorder Research
Share to Neonatal and fetal brain pathology
Share to Neuroinflammation and Neurodegeneration Mechanisms

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 37508346
DOI: 10.3390/biology12070914

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 19 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Autism Spectrum Disorder: A Neuro-Immunometabolic Hypothesis of the Developmental OriginsPMID 37508346Read ↗

Inflammation and the Potential Implication of Macrophage-Microglia Polarization in Human ASD: An OverviewSemantic proximity Read ↗

Etiology of autism spectrum disorders: recent advances and emerging directions.Semantic proximity Read ↗

Microenvironment dysregulation in autism spectrum disorder: Immune, epigenetic, synaptic, and gut-brain mechanisms.Semantic proximity Read ↗

The Energy-Deficit Hypothesis of Autism: Multi-Cytokine Convergence on Mitochondrial Dysfunction: A Cerebral Energy-Deficit Hypothesis of Autism Spectrum DisorderSemantic proximity Read ↗

Risk factors in autism: Thinking outside the brain.Semantic proximity Read ↗

Metabolic and Inflammatory Dysregulation in Autism Spectrum Disorder and COVID-19: A Hypothesis-Generating ReviewSemantic proximity Read ↗

Neurodevelopmental Impact of Maternal Immune Activation and Autoimmune Disorders, Environmental Toxicants and Folate Metabolism on Autism Spectrum DisorderSemantic proximity Read ↗

Expression profiling of autism candidate genes during human brain development implicates central immune signaling pathways.Semantic proximity Read ↗

The early life exposome and autism risk: a role for the maternal microbiome?Semantic proximity Read ↗

Neuroinflammation and Oxidative Stress in the Pathogenesis of Autism Spectrum DisorderSemantic proximity Read ↗

Maternal Immune Activation and Autism in Offspring: What Is the Evidence for Causation?Semantic proximity Read ↗

Neuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and MicrobiotaSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.