Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Ferroptosis hijacking by Mycobacterium tuberculosis

17 Mar 2023

Abstract excerpt

A recent study from Nature Communications reveals that Mycobacterium tuberculosis can hijack epigenetic machinery in host cells and induce host cell ferroptosis, which promotes pathogen pathogenicity and spread. These findings also suggest new therapeutic strategies to treat tuberculosis.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Epigenetics and DNA Methylation
Share to Ferroptosis and cancer prognosis
Share to RNA modifications and cancer

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36932073
DOI: 10.1038/s41467-023-37149-w

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Ferroptosis hijacking by Mycobacterium tuberculosisPMID 36932073Read ↗

Epigenetic Mechanisms Induced by Mycobacterium tuberculosis to Promote Its Survival in the HostSemantic proximity Read ↗

A mycobacterial effector promotes ferroptosis-dependent pathogenicity and disseminationSemantic proximity Read ↗

Regulation of Three Virulence Strategies of Mycobacterium tuberculosis: A Success StorySemantic proximity Read ↗

The Role of Ferroptosis and Cuproptosis in Tuberculosis Pathogenesis: Implications for Therapeutic StrategiesSemantic proximity Read ↗

Tuberculosis: What We Don’t Know Can, and Does, Hurt UsSemantic proximity Read ↗

Mitochondria: Powering the Innate Immune Response to Mycobacterium tuberculosis Infection.Semantic proximity Read ↗

Mycobacterium tuberculosis: An Adaptable Pathogen Associated With Multiple Human DiseasesSemantic proximity Read ↗

The Iron Response of Mycobacterium tuberculosis and Its Implications for Tuberculosis Pathogenesis and Novel TherapeuticsSemantic proximity Read ↗

Mycobacterium tuberculosis : Rewiring host cell signaling to promote infectionSemantic proximity Read ↗

Bioinformatics-led discovery of ferroptosis-associated diagnostic biomarkers and molecule subtypes for tuberculosis patientsSemantic proximity Read ↗

The PPE2 protein of Mycobacterium tuberculosis translocates to host nucleus and inhibits nitric oxide productionSemantic proximity Read ↗

Microbe Profile: Mycobacterium tuberculosis: Humanity's deadly microbial foeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.