Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Enterovirus D68 Infection in Human Primary Airway and Brain Organoids: No Additional Role for Heparan Sulfate Binding for Neurotropism

26 Sept 2022

Abstract excerpt

Recent outbreaks of enterovirus D68, a nonpolio enterovirus, is associated with a serious neurological condition in young children, acute flaccid myelitis (AFM). As there is no antiviral treatment or vaccine available for EV-D68 it is important to better understand how EV-D68 causes AFM and why only recent outbreaks are associated with AFM. We investigated if a change in receptor usage of EV-D68 increases the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to RNA and protein synthesis mechanisms
Share to RNA regulation and disease
Share to Viral Infections and Immunology Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36154279
DOI: 10.1128/spectrum.01694-22

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Enterovirus D68 Infection in Human Primary Airway and Brain Organoids: No Additional Role for Heparan Sulfate Binding for NeurotropismPMID 36154279Read ↗

Enterovirus D68 molecular and cellular biology and pathogenesisSemantic proximity Read ↗

Contemporary Circulating Enterovirus D68 Strains Have Acquired the Capacity for Viral Entry and Replication in Human Neuronal CellsSemantic proximity Read ↗

Enhanced Enterovirus D68 Replication in Neuroblastoma Cells Is Associated with a Cell Culture-Adaptive Amino Acid Substitution in VP1Semantic proximity Read ↗

The pathogenesis and virulence of enterovirus-D68 infectionSemantic proximity Read ↗

In vitro and in vivo models for the study of EV-D68 infectionSemantic proximity Read ↗

Neurotropism of Enterovirus D68 Isolates Is Independent of Sialic Acid and Is Not a Recently Acquired PhenotypeSemantic proximity Read ↗

Contemporary circulating enterovirus D68 strains show differential viral entry and replication in human neuronal cellsSemantic proximity Read ↗

Neurotropism of enterovirus D68 isolates is independent of sialic acid and is not a recently acquired phenotypeSemantic proximity Read ↗

Enterovirus D68 VP1 and VP3 determine neurotropism in human spinal cord organoidsSemantic proximity Read ↗

Enterovirus D68 VP1 and VP3 Determine Neurotropism in Human Spinal Cord Organoids.Semantic proximity Read ↗

Contemporary enterovirus-D68 isolates infect human spinal cord organoidsSemantic proximity Read ↗

Characterization of enterovirus D68 infection in four nonhuman primate speciesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.