Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The maternal-placental-fetal interface: Adaptations of the HPA axis and immune mediators following maternal stress and prenatal alcohol exposure

20 May 2022

Abstract excerpt

This review addresses underlying physiological, cellular, and molecular factors that alter the developing fetal brain stress circuits and responses of the hypothalamic-pituitary-adrenal (HPA) axis caused by maternal stress and prenatal alcohol exposure (PAE). An emphasis is placed on the contribution of the placenta following maternal stress separately, and as a co-occurrence with PAE. Altered fetal HPA axis...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Prenatal Substance Exposure Effects
Share to Stress Responses and Cortisol

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 35605668
DOI: 10.1016/j.expneurol.2022.114121

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The maternal-placental-fetal interface: Adaptations of the HPA axis and immune mediators following maternal stress and prenatal alcohol exposurePMID 35605668Read ↗

Independent and Combined Effects of Prenatal Alcohol Exposure and Prenatal Stress on Fetal HPA Axis DevelopmentSemantic proximity Read ↗

Prenatal maternal stress, fetal programming, and mechanisms underlying later psychopathology—A global perspectiveSemantic proximity Read ↗

Maternal stress, placental 11β-hydroxysteroid dehydrogenase type 2, and infant HPA axis development in humans: Psychosocial and physiological pathwaysSemantic proximity Read ↗

Fetal alcohol spectrum disorders and their transmission through genetic and epigenetic mechanismsSemantic proximity Read ↗

Sex differences in offspring risk and resilience following 11β-hydroxylase antagonism in a rodent model of maternal immune activationSemantic proximity Read ↗

Prenatal maternal stress and offspring aggressive behavior: Intergenerational and transgenerational inheritanceSemantic proximity Read ↗

Effects of maternal exposure to social stress during pregnancy: consequences for mother and offspringSemantic proximity Read ↗

Long‐term Effects of Prenatal Stress and Glucocorticoid ExposureSemantic proximity Read ↗

Prenatal ethanol exposure and changes in fetal neuroendocrine metabolic programmingSemantic proximity Read ↗

Sexual dimorphism in placental development and its contribution to health and diseasesSemantic proximity Read ↗

Dimorphic placental stress: A repercussion of interaction between endocrine disrupting chemicals (EDCs) and fetal sex.Semantic proximity Read ↗

Developmental programming of the HPA axis and related behaviours: epigenetic mechanismsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.