Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Anti‑fibrotic effects and the mechanism of action of miR‑29c in silicosis

22 Feb 2021

Abstract excerpt

Despite increasing evidence suggesting a role for the miR‑29 family in the suppression of fibrosis, its role in silicosis remains unknown. The present study aimed to examine the anti‑fibrotic effects and specific mechanism of action of microRNA (miR)‑29c in pulmonary silicosis using animal and cell models. miR‑29c expression levels were examined in the lungs of silicotic rats via reveres...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
Share to Medical Imaging and Pathology Studies
Share to Occupational and environmental lung diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 33649785
DOI: 10.3892/mmr.2021.11932

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Anti‑fibrotic effects and the mechanism of action of miR‑29c in silicosisPMID 33649785Read ↗

MicroRNA-29b Mediates Lung Mesenchymal-Epithelial Transition and Prevents Lung Fibrosis in the Silicosis ModelSemantic proximity Read ↗

miR-29 Is a Major Regulator of Genes Associated with Pulmonary FibrosisSemantic proximity Read ↗

MicroRNA mimicry blocks pulmonary fibrosisSemantic proximity Read ↗

Role of the microRNA-29 family in fibrotic skin diseasesSemantic proximity Read ↗

Lung fibroblasts express a miR-19a-19b-20a sub-cluster to suppress TGF-β-associated fibroblast activation in murine pulmonary fibrosisSemantic proximity Read ↗

A strategic expression method of miR-29b and its anti-fibrotic effect based on RNA-sequencing analysisSemantic proximity Read ↗

Systemic Delivery of scAAV8-Encoded MiR-29a Ameliorates Hepatic Fibrosis in Carbon Tetrachloride-Treated MiceSemantic proximity Read ↗

Micro-RNA profiling reveals a role for miR-29 in human and murine liver fibrosisSemantic proximity Read ↗

The Antifibrotic Effects and Mechanisms of MicroRNA-26a Action in Idiopathic Pulmonary FibrosisSemantic proximity Read ↗

The Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in Pulmonary FibrosisSemantic proximity Read ↗

The MicroRNA MiR-29c Alleviates Renal Fibrosis via TPM1-Mediated Suppression of the Wnt/β-Catenin PathwaySemantic proximity Read ↗

Differential expression of lncRNAs during silicosis and the role of LOC103691771 in myofibroblast differentiation induced by TGF-β1Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.